Protector turns predator: Autophagic death via selective degradation of KRAS

Autophagy

Volumn 9, Issue 9, 2013, Pages 1438-1439

  Kohli, Latika ^a,b,c   Kaza, Niroop ^a   Carroll, Steven L ^a   Roth, Kevin A ^a  

^a University of Alabama at Birmingham   (United States)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Autophagy; EGFR; KRAS; PRKC; Tamoxifen; Tumor cells

Indexed keywords

EFFECTOR CASPASE; EPIDERMAL GROWTH FACTOR RECEPTOR; ESTROGEN RECEPTOR; MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN KINASE C; TAMOXIFEN;

AUTOPHAGY; CALCIUM CELL LEVEL; CANCER PATIENT; CELL DEATH; CELL KILLING; CYTOTOXICITY; DYNAMICS; HUMAN; MULTIPLE CANCER; OBSERVATIONAL STUDY; OUTCOME ASSESSMENT; PREDATOR; PROTEIN DEGRADATION; PROTEIN PHOSPHORYLATION; PROTEIN TARGETING; SHORT SURVEY; TUMOR CELL; TURNOVER TIME;

AUTOPHAGY; EGFR; KRAS; PRKC; TAMOXIFEN; TUMOR CELLS;

AUTOPHAGY; CYTOPROTECTION; MAP KINASE SIGNALING SYSTEM; PROTEIN KINASE C; PROTEOLYSIS; PROTO-ONCOGENE PROTEINS P21(RAS); TAMOXIFEN;

EID: 84884321046     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.4161/auto.25837     Document Type: Short Survey

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