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Volumn 2, Issue 1, 2013, Pages
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Activation of cGMP/protein kinase G pathway in postconditioned myocardium depends on reduced oxidative stress and preserved endothelial nitric oxide synthase coupling.
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Author keywords
[No Author keywords available]
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Indexed keywords
3 NITROTYROSINE;
3-NITROTYROSINE;
5,6,7,8-TETRAHYDROBIOPTERIN;
BIOPTERIN;
CYCLIC GMP;
CYCLIC GMP DEPENDENT PROTEIN KINASE;
DRUG DERIVATIVE;
ENDOTHELIAL NITRIC OXIDE SYNTHASE;
ENZYME INHIBITOR;
NITRATE;
NITRITE;
NOS3 PROTEIN, RAT;
PHOSPHATIDYLINOSITOL 3 KINASE;
PROTEIN KINASE B;
SUPEROXIDE;
TETRAHYDROBIOPTERIN;
TYROSINE;
ANIMAL;
ARTICLE;
CELL DEATH;
DISEASE MODEL;
DRUG ANTAGONISM;
DRUG EFFECT;
ENZYME ACTIVATION;
ENZYMOLOGY;
HEART INFARCTION;
HEART MUSCLE;
ISCHEMIC POSTCONDITIONING;
MALE;
METABOLISM;
OXIDATIVE STRESS;
PATHOLOGY;
PHOSPHORYLATION;
RAT;
REPERFUSION INJURY;
SIGNAL TRANSDUCTION;
SPRAGUE DAWLEY RAT;
TIME;
ANIMALS;
BIOPTERIN;
CELL DEATH;
CYCLIC GMP;
CYCLIC GMP-DEPENDENT PROTEIN KINASES;
DISEASE MODELS, ANIMAL;
ENZYME ACTIVATION;
ENZYME INHIBITORS;
ISCHEMIC POSTCONDITIONING;
MALE;
MYOCARDIAL INFARCTION;
MYOCARDIAL REPERFUSION INJURY;
MYOCARDIUM;
NITRATES;
NITRIC OXIDE SYNTHASE TYPE III;
NITRITES;
OXIDATIVE STRESS;
PHOSPHATIDYLINOSITOL 3-KINASE;
PHOSPHORYLATION;
PROTO-ONCOGENE PROTEINS C-AKT;
RATS;
RATS, SPRAGUE-DAWLEY;
SIGNAL TRANSDUCTION;
SUPEROXIDES;
TIME FACTORS;
TYROSINE;
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EID: 84884180824
PISSN: None
EISSN: 20479980
Source Type: None
DOI: 10.1161/JAHA.112.005975 Document Type: Article |
Times cited : (60)
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References (0)
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