Phenotype transition of monocytes and liver injury

Liver International

Volumn 33, Issue 9, 2013, Pages 1450-1450

  Zhou, Dexi ^a   Lin, Zhen ^a   Kong, Lingna ^a   Li, Jun ^a  

^a ANHUI MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

CARBON TETRACHLORIDE; CHEMOKINE RECEPTOR CCR; FARNESOID X RECEPTOR; PARACETAMOL;

APOPTOSIS; CELL TYPE; CHOLESTASIS; INFLAMMATION; KUPFFER CELL; LETTER; LIVER INJURY; MACROPHAGE; MONOCYTE; MONONUCLEAR PHAGOCYTE; NEUTROPHIL; NONALCOHOLIC FATTY LIVER; NONHUMAN; PHENOTYPE; STELLATE CELL; TISSUE REPAIR;

ANIMALS; CHOLESTASIS, INTRAHEPATIC; FEMALE; KUPFFER CELLS; MONONUCLEAR PHAGOCYTE SYSTEM;

EID: 84883760799     PISSN: 14783223     EISSN: 14783231     Source Type: Journal    
DOI: 10.1111/liv.12246     Document Type: Letter

References (5)

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2. Holt MP, Cheng L, Ju C. Identification and characterization of infiltrating macrophages in acetaminophen-induced liver injury. J Leukoc Biol 2008; 84: 1410-21.
3. Seki E, De Minicis S, Inokuchi S, et al. CCR2 promotes hepatic fibrosis in mice. Hepatology 2009; 50: 185-97.
4. Chu PS, Nakamoto N, Ebinuma H, et al. C-C motif chemokine receptor 9 positive macrophages activate hepatic stellate cells and promote liver fibrosis in mice. Hepatology 2013; 58: 337-50.
5. Mcmahan R, Wang XX, Cheng LL, et al. Bile-acid receptor activation modulates hepatic monocyte activity and improves non-alcoholic fatty liver disease. J Biol Chem 2013; 288: 11761-70.