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Volumn 54, Issue 5, 2013, Pages 3526-3537
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SIK2 is involved in the negative modulation of insulin-dependent muller cell survival and implicated in hyperglycemia-induced cell death.
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Author keywords
[No Author keywords available]
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Indexed keywords
AKT1 PROTEIN, RAT;
INSULIN;
INSULIN RECEPTOR SUBSTRATE;
IRS1 PROTEIN, RAT;
PROTEIN KINASE B;
PROTEIN SERINE THREONINE KINASE;
SIK2 PROTEIN, RAT;
AGE;
ANIMAL;
ARTICLE;
CELL DEATH;
CELL LINE;
CELL SURVIVAL;
CHRONIC DISEASE;
CHRONIC HYPERGLYCEMIA;
CYTOLOGY;
DIABETIC RETINOPATHY;
EXPERIMENTAL DIABETES MELLITUS;
GENE SILENCING;
GENETICS;
GLIA;
HYPERGLYCEMIA;
MALE;
METABOLISM;
MÜLLER GLIA;
NICK END LABELING;
PATHOLOGY;
PHYSIOLOGY;
RAT;
RETINA;
SIGNAL TRANSDUCTION;
SIK2;
WISTAR RAT;
CHRONIC HYPERGLYCEMIA;
INSULIN;
MÜLLER GLIA;
SIK2;
AGE FACTORS;
ANIMALS;
CELL DEATH;
CELL LINE;
CELL SURVIVAL;
CHRONIC DISEASE;
DIABETES MELLITUS, EXPERIMENTAL;
DIABETIC RETINOPATHY;
GENE SILENCING;
HYPERGLYCEMIA;
IN SITU NICK-END LABELING;
INSULIN;
INSULIN RECEPTOR SUBSTRATE PROTEINS;
MALE;
NEUROGLIA;
PROTEIN-SERINE-THREONINE KINASES;
PROTO-ONCOGENE PROTEINS C-AKT;
RATS;
RATS, WISTAR;
RETINA;
SIGNAL TRANSDUCTION;
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EID: 84883593357
PISSN: None
EISSN: 15525783
Source Type: None
DOI: 10.1167/iovs.12-10729 Document Type: Article |
Times cited : (17)
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References (0)
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