|
|
Volumn 18, Issue 2, 2013, Pages 145-146
|
|
Mitochondrial dysfunction indirectly elevates ROS production by the endoplasmic reticulum
|
|
Author keywords
[No Author keywords available]
|
|
Indexed keywords
BINDING PROTEIN;
CYTOCHROME C OXIDASE;
GUANOSINE TRIPHOSPHATASE;
HYDROGEN PEROXIDE;
MITOCHONDRIAL DNA;
PARKIN;
REACTIVE OXYGEN METABOLITE;
REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE;
REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE;
SUPEROXIDE;
TRANSCRIPTION FACTOR;
TRANSCRIPTION FACTOR IID;
AEROBIC METABOLISM;
ARTICLE;
CELL DAMAGE;
CELL VIABILITY;
ENDOPLASMIC RETICULUM;
ENDOPLASMIC RETICULUM ASSOCIATED DEGRADATION;
MITOCHONDRIAL DYSFUNCTION;
MITOCHONDRIAL RESPIRATION;
MITOCHONDRION;
NONHUMAN;
OUTER MEMBRANE;
PATHOLOGY;
PRIORITY JOURNAL;
PROTEIN METABOLISM;
PROTON MOTIVE FORCE;
RESPIRATORY CHAIN;
SACCHAROMYCES CEREVISIAE;
T LYMPHOCYTE ACTIVATION;
CYTOCHROME-C OXIDASE DEFICIENCY;
ELECTRON TRANSPORT COMPLEX IV;
RAS PROTEINS;
REACTIVE OXYGEN SPECIES;
SACCHAROMYCES CEREVISIAE;
|
|
EID: 84881345531
PISSN: 15504131
EISSN: 19327420
Source Type: Journal
DOI: 10.1016/j.cmet.2013.07.006 Document Type: Short Survey |
|
Times cited : (178)
|
|
References (10)
|
