Naturally occurring autoantibodies interfere with β-amyloid metabolism and improve cognition in a transgenic mouse model of Alzheimer's disease 24 h after single treatment.

Translational psychiatry

Volumn 3, Issue , 2013, Pages

  Mengel, D ^a   Roskam S ^b   Neff, F ^b   Balakrishnan, K ^b   Deuster, O ^b   Gold, M ^b   Oertel, W H ^b   Bacher, M ^b   Bach, J P ^b   Dodel, R ^b  

^a PHILIPPS UNIVERSITY MARBURG   (Germany)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; AUTOANTIBODY; CYTOKINE;

ALZHEIMER DISEASE; ANIMAL; ARTICLE; COGNITION; DISEASE MODEL; ENZYME LINKED IMMUNOSORBENT ASSAY; FEMALE; METABOLISM; METHODOLOGY; MOUSE; PHYSIOLOGY; TRANSGENIC ANIMAL; TRANSGENIC MOUSE; WESTERN BLOTTING;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; ANIMALS, GENETICALLY MODIFIED; AUTOANTIBODIES; BLOTTING, WESTERN; COGNITION; CYTOKINES; DISEASE MODELS, ANIMAL; ENZYME-LINKED IMMUNOSORBENT ASSAY; FEMALE; MICE; MICE, TRANSGENIC;

EID: 84881340502     PISSN: None     EISSN: 21583188     Source Type: None    
DOI: 10.1038/tp.2012.151     Document Type: Article

References (0)