Why Are Sensory Axons More Vulnerable for Ischemia than Motor Axons?

PLoS ONE

Volumn 8, Issue 6, 2013, Pages

  Hofmeijer, Jeannette ^a,b   Franssen, Hessel ^c   van Schelven, Leonard J ^c   van Putten, Michel J A M ^a,d  

^a UNIVERSITY OF TWENTE   (Netherlands)

^b RIJNSTATE HOSPITAL   (Netherlands)

^c UNIVERSITY MEDICAL CENTER UTRECHT   (Netherlands)

^d MEDISCH SPECTRUM TWENTE   (Netherlands)

Author keywords

[No Author keywords available]

Indexed keywords

ADULT; ARTICLE; CELL ACTIVITY; COMPOUND MUSCLE ACTION POTENTIAL; COMPOUND SENSORY ACTION POTENTIAL; ENZYME INACTIVATION; FEMALE; FOREARM ISCHEMIA; HUMAN; HUMAN EXPERIMENT; ISCHEMIA; MALE; MOTONEURON; NERVE CELL MEMBRANE STEADY POTENTIAL; NERVE CONDUCTION; NERVE EXCITABILITY; NERVE FUNCTION; NERVE POTENTIAL; NERVOUS SYSTEM PARAMETERS; NORMAL HUMAN; PERIPHERAL NEUROPATHY; REFRACTORY PERIOD; SENSORY NERVE CELL; SODIUM CONDUCTANCE; STRENGTH DURATION TIME CONSTANT; SYMPTOMATOLOGY;

ACTION POTENTIALS; ADULT; ARM; AXONS; CELL HYPOXIA; FEMALE; HUMANS; ISCHEMIA; MALE; MIDDLE AGED; MOTOR NEURONS; NEURAL CONDUCTION; PROPRIOCEPTION; SENSORY RECEPTOR CELLS;

EID: 84879267735     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0067113     Document Type: Article

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