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Medical Principles and Practice
Volumn 22, Issue 4, 2013, Pages 311-312
What's new in the pathophysiology of sickle cell disease?
Author keywords

[No Author keywords available]
Indexed keywords

AMINO ACID; BETA GLOBULIN; GLUTAMIC ACID; HEMOGLOBIN S; NITRIC OXIDE; VALINE;
EID: 84879125504     PISSN: 10117571     EISSN: 14230151     Source Type: Journal    
DOI: 10.1159/000350283     Document Type: Short Survey
Times cited : (15)
References (10)
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  • 3
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    • Eaton, W.R.1    Hofrichter, J.2
  • 4
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    • Current perspectives on the kinetics of hemoglobin S gelation
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    • San Biagio, P.L.1    Hofrichter, J.2    Mozzarelli, A.3
  • 5
    • 69549107429 scopus 로고    scopus 로고
    • Vasculopathy in sickle cell disease: Biology, pathophysiology, genetics, translational medicine, and new research directions
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    • Kato, G.J.1    Hebbel, R.P.2    Steinberg, M.H.3
  • 6
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    • Mechanisms of vasculopathy in sickle cell disease and thalassemia
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    • (2008) Hematology Am Soc Hematology Educ Program , pp. 177-185
    • Morris, C.R.1
  • 7
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    • Vascular risk assessment in patients with sickle cell disease
    • Morris CR: Vascular risk assessment in patients with sickle cell disease. Haematologica 2011; 96: 1-5.
    • (2011) Haematologica , vol.96 , pp. 1-5
    • Morris, C.R.1
  • 8
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    • Arginase: A critical regulator of nitric oxide synthesis and vascular function
    • Durante W, Johnson FK, Johnson RA: Arginase: a critical regulator of nitric oxide synthesis and vascular function. Clin Exp Pharmacol Physiol 2007; 34: 906-911.
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  • 9
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    • Pulmonary hypertension as a risk factor for death in patients with sickle cell disease
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    • Genetic modifiers of sickle cell disease
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.