BLNK GENE;
CBL GENE;
CD79B GENE;
GENE IDENTIFICATION;
HUMAN;
LETTER;
MOLECULAR PATHOLOGY;
MUTATIONAL ANALYSIS;
MUTATOR GENE;
PRIMARY CENTRAL NERVOUS SYSTEM LYMPHOMA;
PRIORITY JOURNAL;
SHIP GENE;
SIGNAL TRANSDUCTION;
B-LYMPHOCYTES;
CENTRAL NERVOUS SYSTEM;
CENTRAL NERVOUS SYSTEM NEOPLASMS;
GENE EXPRESSION REGULATION, NEOPLASTIC;
HUMANS;
LYMPHOMA;
MUTATION;
RECEPTORS, ANTIGEN, B-CELL;
SIGNAL TRANSDUCTION;
EID: 84878372110PISSN: 00016322EISSN: 14320533Source Type: Journal DOI: 10.1007/s00401-012-1064-7Document Type: Letter
Activating L265P mutations of the MYD88 gene are common in primary central nervous system lymphoma
22020631 10.1007/s00401-011-0891-2
Montesinos-Rongen M, Godlewska E, Brunn A, Wiestler OD, Siebert R, Deckert M (2011) Activating L265P mutations of the MYD88 gene are common in primary central nervous system lymphoma. Acta Neuropathol 122:791-792
Montesinos-Rongen M, Küppers R, Schlüter D et al (1999) Primary central nervous system lymphomas are derived from germinal-center B cells and show a preferential usage of the V4-34 gene segment. Am J Pathol 155:2077-2086
Montesinos-Rongen M, Schmitz R, Brunn A et al (2010) Mutations of CARD11 but not TNFAIP3 may activate the NF-kappaB pathway in primary CNS lymphoma. Acta Neuropathol 120:529-535
Montesinos-Rongen M, Van Roost D, Schaller C, Wiestler OD, Deckert M (2004) Primary diffuse large B-cell lymphomas of the central nervous system are targeted by aberrant somatic hypermutation. Blood 103:1869-1875
