Nutrient-driven O-GlcNAc cycling influences autophagic flux and neurodegenerative proteotoxicity

Autophagy

Volumn 9, Issue 4, 2013, Pages 604-606

  Wang, Peng ^a   Hanover, John A ^a  

^a NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES   (United States)

Author keywords

Autophagy; Insulin signaling; Neurodegeneration; O GlcNAc; Proteasome; Proteostasis; Proteotoxicity

Indexed keywords

AMYLOID BETA PROTEIN; INSULIN; O LINKED GLCNAC TRANSFERASE; PHOSPHATIDYLETHANOLAMINE; POLYGLUTAMINE; PROTEASOME; PROTEIN BCL 2; TRANSFERASE; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; AUTOPHAGOSOME; AUTOPHAGY; CAENORHABDITIS ELEGANS; ENZYME ACTIVITY; ENZYME METABOLISM; ENZYME SYNTHESIS; LOSS OF FUNCTION MUTATION; NERVE DEGENERATION; PHAGOSOME; PROTEIN HOMEOSTASIS; PROTEIN PHOSPHORYLATION; PROTEOTOXICITY; TAUOPATHY; TOXICITY;

EID: 84877358993     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.4161/auto.23459     Document Type: Article

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