Improvement of ER stress-induced diabetes by stimulating autophagy

Autophagy

Volumn 9, Issue 4, 2013, Pages 626-628

  Bachar Wikstrom, Etty ^a   Wikstrom, Jakob D ^a   Kaiser, Nurit ^a   Cerasi, Erol ^a   Leibowitz, Gil ^a  

^a HADASSAH HEBREW UNIVERSITY MEDICAL CENTER   (Israel)

Author keywords

Autophagy Abbreviations; Beta cell; Diabetes; Endoplasmic reticulum; Endoplasmic reticulum associated degradation; ER; ER stress; ERAD; IAPP; Islet amyloid polypeptide; Mechanistic target of rapamycin complex 1; MIDY; MTOR; MTORC1; Mutant INS gene induced diabetes of youth syndrome; Rapamycin

Indexed keywords

CHAPERONE; MAMMALIAN TARGET OF RAPAMYCIN COMPLEX 1; PROINSULIN; RAPAMYCIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; AUTOPHAGOSOME; AUTOPHAGY; CELL PROTECTION; CELL STIMULATION; CELL STRESS; CONTROLLED STUDY; DIABETES MELLITUS; DISULFIDE BOND; ENDOPLASMIC RETICULUM STRESS; GENE MUTATION; LYSOSOME; MOLECULAR INTERACTION; MOUSE; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PANCREAS ISLET BETA CELL; PROTEIN FOLDING; UNFOLDED PROTEIN RESPONSE;

EID: 84877315340     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.4161/auto.23642     Document Type: Article

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