From GWAS risk foci to glioma molecular subclass

Neuro-Oncology

Volumn 15, Issue 5, 2013, Pages 513-514

  Yung, W K Alfred ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIN DEPENDENT KINASE INHIBITOR 2B; EPIDERMAL GROWTH FACTOR RECEPTOR; ISOCITRATE DEHYDROGENASE; PROTEIN P53;

CANCER PROGNOSIS; EDITORIAL; GENE LOCUS; GENE MUTATION; GENETIC ASSOCIATION; GENETIC PREDISPOSITION; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENETIC VARIABILITY; GENOTYPE PHENOTYPE CORRELATION; GLIOMA; HETEROZYGOTE; HISTOPATHOLOGY; HUMAN; MOLECULAR MECHANICS; PATHOGENESIS; SINGLE NUCLEOTIDE POLYMORPHISM;

BRAIN NEOPLASMS; CHROMOSOMES, HUMAN; CHROMOSOMES, HUMAN, PAIR 11; FEMALE; GENETIC PREDISPOSITION TO DISEASE; GLIOMA; HUMANS; ISOCITRATE DEHYDROGENASE; MALE; MUTATION; POLYMORPHISM, SINGLE NUCLEOTIDE; TUMOR MARKERS, BIOLOGICAL;

EID: 84877125719     PISSN: 15228517     EISSN: 15235866     Source Type: Journal    
DOI: 10.1093/neuonc/not061     Document Type: Editorial

References (5)

1. Walsh KM, Anderson E, HansenHM, et al. Analysis of 60 reported glioma risk SNPs replicates published GWAS findings but fails to replicate associations from published candidate-gene studies. Genet Epidemiol. 2013; 37(2): 222-228.
2. Jenkins RB, Wrensch MR, Johnson D, et al. Distinct germ line polymorphisms underlie glioma morphologic heterogeneity. Cancer Genet. 2011; 204(1): 13-18.
3. SimonM, Hosking FJ, Marie Y, et al. Genetic risk profiles identify different molecular etiologies for glioma. Clin Cancer Res.2010; 16(21): 5252-5259.
4. Rice T, Zheng S, Decker PA, et al. Inherited variant on chromosome 11q23 increases susceptibility to IDH-mutated but not IDH-normal gliomas regardless of grade or histology. Neuro Oncol. 2013; 15(5): 535-541.
5. Di Stefano AL, Enciso-Mora V, Marie Y, et al. Association between glioma susceptibility loci and tumor pathology defines specific molecular etiologies. Neuro Oncol. 2013; 15(5): 542-547.