Biological mechanisms underlying neuroprogression in bipolar disorder

Revista Brasileira de Psiquiatria

Volumn 35, Issue 1, 2013, Pages 1-2

  Budni, Josiane ^a   Valvassori, Samira S ^a   Quevedo, João ^a  

^a UNIVERSIDADE DO EXTREMO SUL CATARINENSE   (Brazil)

Author keywords

[No Author keywords available]

Indexed keywords

BRAIN DERIVED NEUROTROPHIC FACTOR; INTERLEUKIN 1BETA; INTERLEUKIN 6; LITHIUM; MESSENGER RNA; NEUROLEPTIC AGENT; PROTEIN BAX; TUMOR NECROSIS FACTOR ALPHA; VALPROIC ACID;

APOPTOSIS; BIPOLAR DISORDER; COGNITIVE DEFECT; COMORBIDITY; DISEASE ASSOCIATION; DISEASE SEVERITY; EDITORIAL; ENERGY METABOLISM; FUNCTIONAL DISEASE; HUMAN; INFLAMMATION; NEUROBIOLOGY; NEUROTROPISM; OXIDATIVE STRESS; PHENOMENOLOGY; TREATMENT RESPONSE;

EID: 84876164795     PISSN: 15164446     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.rbp.2013.01.002     Document Type: Editorial

References (5)

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2. Kim HW, Rapoport SI, Rao JS. Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients. Neurobiol Dis. 2010;37:596-603.
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4. Thompson Ray M, Weickert CS, Wyatt E, Webster MJ. Decreased BDNF, trkB-TK+ and GAD67 mRNA expression in the hippocampus of individuals with schizophrenia and mood disorders. J Psychiatry Neurosci. 2011;36:195-203.
5. Kapczinski F, Frey BN, Andreazza AC, Kauer-Sant'Anna M, Cunha AB, Post RM. Increased oxidative stress as a mechanism for decreased BDNF levels in acute manic episodes. Rev Bras Psiquiatr. 2008;30:243-5.