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Volumn 153, Issue 6, 2012, Pages 825-827
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Energy deficit as a possible factor for the induction of caspase-dependent apoptosis in left ventricular myocardial cells during genetically determined and secondary arterial hypertension
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Author keywords
Apoptosis; Arterial hypertension; Caspase; Myocardium; Phosphocreatine
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Indexed keywords
ADENOSINE TRIPHOSPHATE;
CASPASE 3;
CASPASE 8;
CREATINE PHOSPHATE;
CARDIOTONIC AGENT;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
APOPTOSIS;
ARTICLE;
CELL ENERGY;
CELLULAR, SUBCELLULAR AND MOLECULAR BIOLOGICAL PHENOMENA AND FUNCTIONS;
CONTROLLED STUDY;
ENERGY DEFICIT;
ENERGY METABOLISM;
ENZYME ACTIVITY;
ENZYME ANALYSIS;
HEART MUSCLE CELL;
INTRACELLULAR SIGNALING;
MALE;
MITOCHONDRIAL RESPIRATION;
MOLECULAR PATHOLOGY;
NONHUMAN;
RABBIT;
RAT;
RENOVASCULAR HYPERTENSION;
ANIMAL;
CHEMISTRY;
DRUG EFFECT;
GENE EXPRESSION;
GENETICS;
HEART MUSCLE;
HEART VENTRICLE;
METABOLISM;
PATHOPHYSIOLOGY;
SPONTANEOUSLY HYPERTENSIVE RAT;
WISTAR KYOTO RAT;
ORYCTOLAGUS CUNICULUS;
RATTUS;
ANIMALS;
APOPTOSIS;
CARDIOTONIC AGENTS;
CASPASE 3;
CASPASE 8;
ENERGY METABOLISM;
GENE EXPRESSION;
HEART VENTRICLES;
HYPERTENSION, RENOVASCULAR;
MALE;
MYOCARDIUM;
PHOSPHOCREATINE;
RABBITS;
RATS;
RATS, INBRED SHR;
RATS, INBRED WKY;
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EID: 84875606394
PISSN: 00074888
EISSN: 15738221
Source Type: Journal
DOI: 10.1007/s10517-012-1835-0 Document Type: Article |
Times cited : (1)
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References (11)
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