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Volumn 19, Issue 1, 2013, Pages 26-27

Unlocking the mysterious mechanisms of myc

Author keywords

[No Author keywords available]

Indexed keywords

MYC PROTEIN;

EID: 84874576085     PISSN: 10788956     EISSN: 1546170X     Source Type: Journal    
DOI: 10.1038/nm.3060     Document Type: Article
Times cited : (10)

References (15)
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    • Lin, C.Y.1
  • 2
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    • C-Myc is a universal amplifier of expressed genes in lymphocytes and embryonic stem cells
    • Nie, Z. et al. c-Myc is a universal amplifier of expressed genes in lymphocytes and embryonic stem cells. Cell 151, 68–79 (2012).
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    • Nie, Z.1
  • 3
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    • TGFβ influences Myc, Miz-1 and Smad to control the CDK inhibitor p15INK4b
    • Seoane, J. et al. TGFβ influences Myc, Miz-1 and Smad to control the CDK inhibitor p15INK4b. Nat. Cell Biol. 3, 400–408 (2001).
    • (2001) Nat. Cell Biol , vol.3 , pp. 400-408
    • Seoane, J.1
  • 4
    • 0034744957 scopus 로고    scopus 로고
    • Repression of p15INK4b expression by Myc through association with Miz-1
    • Staller, P. et al. Repression of p15INK4b expression by Myc through association with Miz-1. Nat. Cell Biol. 3, 392–399 (2001).
    • (2001) Nat. Cell Biol , vol.3 , pp. 392-399
    • Staller, P.1
  • 5
    • 77953917870 scopus 로고    scopus 로고
    • The interaction between Myc and Miz1 is required to antagonize TGFβ-dependent autocrine signaling during lymphoma formation and maintenance
    • van Riggelen, J. et al. The interaction between Myc and Miz1 is required to antagonize TGFβ-dependent autocrine signaling during lymphoma formation and maintenance. Genes Dev. 24, 1281–1294 (2010).
    • (2010) Genes Dev , vol.24 , pp. 1281-1294
    • Van Riggelen, J.1
  • 6
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    • Skin epidermis lacking the c-Myc gene is resistant to Ras-driven tumorigenesis but can reacquire sensitivity upon additional loss of the p21Cip1 gene
    • Oskarsson, T. et al. Skin epidermis lacking the c-Myc gene is resistant to Ras-driven tumorigenesis but can reacquire sensitivity upon additional loss of the p21Cip1 gene. Genes Dev. 20, 2024–2029 (2006).
    • (2006) Genes Dev , vol.20 , pp. 2024-2029
    • Oskarsson, T.1
  • 7
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    • MYC suppresses cancer metastasis by direct transcriptional silencing of αv and β3 integrin subunits
    • Liu, H. et al. MYC suppresses cancer metastasis by direct transcriptional silencing of αv and β3 integrin subunits. Nat. Cell Biol. 14, 567–574 (2012).
    • (2012) Nat. Cell Biol , vol.14 , pp. 567-574
    • Liu, H.1
  • 8
    • 29944444852 scopus 로고    scopus 로고
    • Myc regulates keratinocyte adhesion and differentiation via complex formation with Miz1
    • Gebhardt, A. et al. Myc regulates keratinocyte adhesion and differentiation via complex formation with Miz1. J. Cell Biol. 172, 139–149 (2006).
    • (2006) J. Cell Biol , vol.172 , pp. 139-149
    • Gebhardt, A.1
  • 9
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    • c-Myc regulates transcriptional pause release
    • Rahl
    • Rahl, P.B. et al. c-Myc regulates transcriptional pause release. Cell 141, 432–445 (2010).
    • (2010) Cell , vol.141 , pp. 432-445
  • 10
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    • Myc’s broad reach
    • Eilers, M. & Eisenman, R.N. Myc’s broad reach. Genes Dev. 22, 2755–2766 (2008).
    • (2008) Genes Dev , vol.22 , pp. 2755-2766
    • Eilers, M.1    Eisenman, R.N.2
  • 11
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    • Myc, Mondo, and Metabolism
    • Sloan, E.J. & Ayer, D.E. Myc, Mondo, and Metabolism. Genes Cancer 1, 587–596 (2010).
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  • 12
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    • Revisiting global gene expression analysis
    • Lovén, J. et al. Revisiting global gene expression analysis. Cell 151, 476–482 (2012).
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  • 13
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    • Identification of CDK4 as a target of c-MYC
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  • 14
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    • A strategy for identifying transcription factor binding sites reveals two classes of genomic c-Myc target sites
    • Haggerty, T.J. et al. A strategy for identifying transcription factor binding sites reveals two classes of genomic c-Myc target sites. Proc. Natl. Acad. Sci. USA 100, 5313–5318 (2003).
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    • Haggerty, T.J.1
  • 15
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    • Cell-type specific and combinatorial usage of diverse transcription factors revealed by genome-wide binding studies in multiple human cells
    • Lee, B.K. et al. Cell-type specific and combinatorial usage of diverse transcription factors revealed by genome-wide binding studies in multiple human cells. Genome Res. 22, 9–24 (2012).
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    • Lee, B.K.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.