Autophagy activation ameliorates neuronal pathogenesis of FTLD-U mice A new light for treatment of TARDBP/TDP-43 proteinopathies

Autophagy

Volumn 9, Issue 2, 2013, Pages 239-240

  Wang, I Fang ^a,b,c   Tsai, Kuen Jer ^c   Shen, Che Kun James ^a,b  

^a NATIONAL DEFENSE MEDICAL CENTER   (Taiwan)

^b INSTITUTE OF MOLECULAR BIOLOGY   (Taiwan)

^c NATIONAL CHENG KUNG UNIVERSITY   (Taiwan)

Author keywords

Autophagy; FTLD U; Neurodegeneration; Rapamycin; TDP 43

Indexed keywords

CARBAMAZEPINE; MAMMALIAN TARGET OF RAPAMYCIN; RAPAMYCIN; SPERMIDINE; TAMOXIFEN; TAR DNA BINDING PROTEIN;

ALZHEIMER DISEASE; ANIMAL MODEL; AUTOLYSOSOME; AUTOPHAGOSOME; AUTOPHAGY; DEGENERATIVE DISEASE; DISORDERS OF AMINO ACID AND PROTEIN METABOLISM; DRUG EFFICACY; DRUG RESPONSE; FRONTOTEMPORAL DEMENTIA; MOTOR DYSFUNCTION; NERVE DEGENERATION; NEUROPROTECTION; NONHUMAN; NOTE; PROTEINOPATHY; SIGNAL TRANSDUCTION; TDP 43 PROTEINOPATHY;

EID: 84873628557     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.4161/auto.22526     Document Type: Note

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