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Volumn 18, Issue , 2012, Pages 992-1002
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Inhibition of α-SMA by the ectodomain of FGFR2c attenuates lung fibrosis.
a b b b b b b b b b
a
Guangdong Provincial Key Laboratory of Bioengineering Medicine (National Engineering Research Centre of Genetic Medicine)
(China)
b
NONE
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Author keywords
[No Author keywords available]
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Indexed keywords
ACTIN;
ALPHA SMOOTH MUSCLE ACTIN, MOUSE;
ALPHA-SMOOTH MUSCLE ACTIN, MOUSE;
BLEOMYCIN;
FIBROBLAST GROWTH FACTOR 2;
FIBROBLAST GROWTH FACTOR RECEPTOR 2;
ISOPROTEIN;
SMAD PROTEIN;
TRANSFORMING GROWTH FACTOR BETA1;
ANIMAL;
ARTICLE;
C57BL MOUSE;
CELL CULTURE;
CELL PROLIFERATION;
CHEMISTRY;
DISEASE MODEL;
DRUG ANTAGONISM;
DRUG EFFECT;
ENZYME ACTIVATION;
ENZYMOLOGY;
FIBROBLAST;
GENE EXPRESSION REGULATION;
GENETICS;
HUMAN;
LUNG FIBROSIS;
MALE;
METABOLISM;
MOUSE;
PATHOLOGY;
PNEUMONIA;
PROTEIN BINDING;
PROTEIN TERTIARY STRUCTURE;
SIGNAL TRANSDUCTION;
SURFACE PLASMON RESONANCE;
ACTINS;
ANIMALS;
BLEOMYCIN;
CELL PROLIFERATION;
CELLS, CULTURED;
DISEASE MODELS, ANIMAL;
ENZYME ACTIVATION;
FIBROBLAST GROWTH FACTOR 2;
FIBROBLASTS;
GENE EXPRESSION REGULATION;
HUMANS;
MALE;
MAP KINASE SIGNALING SYSTEM;
MICE;
MICE, INBRED C57BL;
PNEUMONIA;
PROTEIN BINDING;
PROTEIN ISOFORMS;
PROTEIN STRUCTURE, TERTIARY;
PULMONARY FIBROSIS;
RECEPTOR, FIBROBLAST GROWTH FACTOR, TYPE 2;
SMAD PROTEINS;
SURFACE PLASMON RESONANCE;
TRANSFORMING GROWTH FACTOR BETA1;
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EID: 84873284266
PISSN: None
EISSN: 15283658
Source Type: Journal
DOI: 10.2119/molmed.2011.00425 Document Type: Article |
Times cited : (44)
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References (0)
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