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Rheumatology International
Volumn 33, Issue 1, 2013, Pages 271-272
The role of interleukin-23/interleukin-17 axis in the development of systemic lupus erythematosus among patients with tuberculosis: Comment on: Lin YC, Liang SJ, Liu YH et al.: Tuberculosis as a risk factor for systemic lupus erythematosus: Results of a nationwide study in Taiwan. (Rheumatol Int. 2011 Mar 18. [Epub ahead of print])
Author keywords

[No Author keywords available]
Indexed keywords

GAMMA INTERFERON; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 17; INTERLEUKIN 23; TRANSCRIPTION FACTOR TWIST; TRANSCRIPTION FACTOR TWIST 1; UNCLASSIFIED DRUG;
EID: 84872339211     PISSN: 01728172     EISSN: 1437160X     Source Type: Journal    
DOI: 10.1007/s00296-011-2226-y     Document Type: Letter
Times cited : (1)
References (7)
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    • (2011) Rheumatol Int
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    • Doreau A, Belot A, Bastid J et al (2009) Interleukin 17 acts in synergy with B cell-activating factor to influence B cell biology and the pathophysiology of systemic lupus erythematosus. Nat Immunol 10:778-785
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    • Wong CK, Lit LC, Tam LS et al (2008) Hyperproduction of IL-23 and IL-17 in patients with systemic lupus erythematosus: implications for Th17-mediated inflammation in auto-immunity. Clin Immunol 127:385-393
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.