Integrin α5β1-fimbriae binding and actin rearrangement are essential for Porphyromonas gingivalis invasion of osteoblasts and subsequent activation of the JNK pathway.

BMC microbiology

Volumn 13, Issue , 2013, Pages

  Zhang, Wenjian ^a   Ju, Jun ^b   Rigney, Todd ^b   Tribble, Gena ^b  

^a UNIVERSITY OF TEXAS DENTAL BRANCH AT HOUSTON   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ACTIN; VERY LATE ACTIVATION ANTIGEN 5;

ANIMAL; APOPTOSIS; ARTICLE; CELL CULTURE; ENDOCYTOSIS; FIMBRIA; HOST PATHOGEN INTERACTION; METABOLISM; MICROBIOLOGY; MOUSE; OSTEOBLAST; PATHOGENICITY; PORPHYROMONAS GINGIVALIS; PROTEIN BINDING; SIGNAL TRANSDUCTION;

ACTINS; ANIMALS; APOPTOSIS; CELLS, CULTURED; ENDOCYTOSIS; FIMBRIAE, BACTERIAL; HOST-PATHOGEN INTERACTIONS; INTEGRIN ALPHA5BETA1; MAP KINASE SIGNALING SYSTEM; MICE; OSTEOBLASTS; PORPHYROMONAS GINGIVALIS; PROTEIN BINDING;

EID: 84872107011     PISSN: None     EISSN: 14712180     Source Type: None    
DOI: 10.1186/1471-2180-13-5     Document Type: Article

References (0)