Silencing of Fas, Fas-associated via death domain, or caspase 3 differentially affects lung inflammation, apoptosis, and development of trauma-induced septic acute lung injury

Shock

Volumn 39, Issue 1, 2013, Pages 19-27

  Messer, Mirko Philipp ^a   Kellermann, Philipp ^a   Weber, Sascha Jörn ^a   Hohmann, Christoph ^a   Denk, Stephanie ^a   Klohs, Bettina ^a   Schultze, Anke ^a   Braumüller, Sonja ^a   Huber Lang, Markus Stefan ^a   Perl, Mario ^a  

^a UNIVERSITY OF ULM   (Germany)

Author keywords

apoptosis; ARDS; cytokines; inflammation; Lung contusion; sepsis; siRNA; trauma

Indexed keywords

CASPASE 3; FAS ANTIGEN; FAS ASSOCIATED DEATH DOMAIN PROTEIN; SMALL INTERFERING RNA;

ACUTE LUNG INJURY; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BLUNT TRAUMA; CONTROLLED STUDY; GENE SILENCING; IMMUNOHISTOCHEMISTRY; LUNG APOPTOSIS; LUNG LAVAGE; LUNG PARENCHYMA; MALE; MOUSE; NEUTROPHIL CHEMOTAXIS; NICK END LABELING; NONHUMAN; PLASMA; PNEUMONIA; POLYMICROBIAL SEPSIS; PROTEIN ANALYSIS; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SEPSIS; THORAX INJURY; WESTERN BLOTTING;

ACUTE LUNG INJURY; ANIMALS; ANTIGENS, CD95; APOPTOSIS; CASPASE 3; CYTOKINES; DISEASE MODELS, ANIMAL; FAS LIGAND PROTEIN; FAS-ASSOCIATED DEATH DOMAIN PROTEIN; GENE SILENCING; GENETIC THERAPY; INTERLEUKIN-10; MALE; MICE; MICE, INBRED C57BL; NEUTROPHIL INFILTRATION; PNEUMONIA; RNA, SMALL INTERFERING; SIGNAL TRANSDUCTION; WOUNDS, NONPENETRATING;

EID: 84871998329     PISSN: 10732322     EISSN: 15400514     Source Type: Journal    
DOI: 10.1097/SHK.0b013e318277d856     Document Type: Article

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