Effect of selenium on proliferation and apoptosis of kaschin-beck disease chondrocyte cultured in vitro

Chinese Journal of Endemiology

Volumn 29, Issue 5, 2010, Pages 480-484

  Duan, Chen ^a   Guo, Xiong ^a   Zhang, Xiao Dong ^a   Gao, Zong Qiang ^a   Zhang, Yin Gang ^a   Yu, Yue Xiang ^a  

^a XI'AN JIAOTONG UNIVERSITY   (China)

Author keywords

Apoptosis; Chondrocytes; Kaschin Beck disease; Selenium

Indexed keywords

CASPASE 3; SELENIUM; TETRAZOLIUM;

ACCIDENT; APOPTOSIS; ARTICULAR CARTILAGE; CARTILAGE CELL; CELL GROWTH; CELL PROLIFERATION; CONTROL GROUP; DIAGNOSIS; HUMAN; IMMUNOCYTOCHEMISTRY; IN VITRO STUDY; KASHIN BECK DISEASE; PATIENT; SUPPLEMENTATION;

EID: 84871601705     PISSN: 10004955     EISSN: None     Source Type: Journal    
DOI: 10.3760/cma.j.issn.1000-4955.2010.05.003     Document Type: Article

References (4)

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2. Wang SJ, Guo X, Zuo H, et al. Chondrocyte apoptosis and expression of Bcl-2, Bax, Fas, and iNOS in articular cartilage in patients with Kaschin-Beck disease [J]. J Rheumatol, 2006, 33(3):615-619. (Pubitemid 43334588)
3. Chen J, Chu Y, Cao J, et al. T-2 toxin induces apoptosis, and selenium partly blocks, T-2 toxin induced apoptosis in chondrocytes through modulation of the Bax/Bcl-2 ratio [J]. Food Chem Toxicol, 2006, 44(4):567-573. (Pubitemid 43375135)
4. Li SY, Cao JL, Shi ZL, et al. Promotion of the articular cartilage proteoglycan degradation by T-2 toxin and selenium protective effect[J]. J Zhejiang Univ Sci B, 2008, 9(1):22-33.