메뉴 건너뛰기




Volumn 30, Issue 4, 2012, Pages 200-205

Imatinib plasma trough levels in chronic myeloid leukaemia: Results of a multicentre study CSTI571AIL11TGLIVEC

Author keywords

Adherence; Chronic myeloid leukaemia; Imatinib

Indexed keywords

IMATINIB;

EID: 84870931711     PISSN: 02780232     EISSN: 10991069     Source Type: Journal    
DOI: 10.1002/hon.2005     Document Type: Article
Times cited : (36)

References (22)
  • 1
    • 68549097002 scopus 로고    scopus 로고
    • International randomized study of interferon versus STI571 (IRIS) 7-year follow-up: sustained survival, low rate of transformation and increased rate of major molecular response (MMR) in patients (pts) with newly diagnosed chronic myeloid leukemia in chronic phase (CML-CP) treated with imatinib (IM)
    • abstract 186].
    • O'Brien SG, Guilhot F, Goldman JM, et al. International randomized study of interferon versus STI571 (IRIS) 7-year follow-up: sustained survival, low rate of transformation and increased rate of major molecular response (MMR) in patients (pts) with newly diagnosed chronic myeloid leukemia in chronic phase (CML-CP) treated with imatinib (IM). Blood 2008; 112(11): 76[abstract 186].
    • (2008) Blood , vol.112 , Issue.11 , pp. 76
    • O'Brien, S.G.1    Guilhot, F.2    Goldman, J.M.3
  • 2
    • 77949767505 scopus 로고    scopus 로고
    • International randomized study of interferon vs STI571 (IRIS) 8-year follow up: sustained survival and low risk for progression or events in patients with newly diagnosed chronic myeloid leukemia in chronic phase (CML-CP) treated with imatinib
    • abstract 1126].
    • Deininger M, O'Brien SG, Guilhot F, et al. International randomized study of interferon vs STI571 (IRIS) 8-year follow up: sustained survival and low risk for progression or events in patients with newly diagnosed chronic myeloid leukemia in chronic phase (CML-CP) treated with imatinib. Blood 2009; 114(22): 462[abstract 1126].
    • (2009) Blood , vol.114 , Issue.22 , pp. 462
    • Deininger, M.1    O'Brien, S.G.2    Guilhot, F.3
  • 3
    • 73349122639 scopus 로고    scopus 로고
    • Chronic myeloid leukemia: an update of concepts and management recommendations of european LeukemiaNet
    • Baccarani M, Cortes J, Pane F, et al. Chronic myeloid leukemia: an update of concepts and management recommendations of european LeukemiaNet. J Clin Oncol 2009; 27(35): 6041-6051.
    • (2009) J Clin Oncol , vol.27 , Issue.35 , pp. 6041-6051
    • Baccarani, M.1    Cortes, J.2    Pane, F.3
  • 4
    • 0035800507 scopus 로고    scopus 로고
    • Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification
    • Gorre ME, Mohammed M, Ellwood K, et al. Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification. Science 2001; 293(5531): 876-880.
    • (2001) Science , vol.293 , Issue.5531 , pp. 876-880
    • Gorre, M.E.1    Mohammed, M.2    Ellwood, K.3
  • 5
    • 77952467377 scopus 로고    scopus 로고
    • Adherence is the critical factor for achieving molecular responses in patients with chronic myeloid leukemia who achieve complete cytogenetic responses on imatinib
    • Marin D, Bazeos A, Mahon FX, et al. Adherence is the critical factor for achieving molecular responses in patients with chronic myeloid leukemia who achieve complete cytogenetic responses on imatinib. J Clin Oncol 2010; 28: 2381-2388.
    • (2010) J Clin Oncol , vol.28 , pp. 2381-2388
    • Marin, D.1    Bazeos, A.2    Mahon, F.X.3
  • 6
    • 1542608328 scopus 로고    scopus 로고
    • Pharmacokinetics and pharmacodynamics of imatinib in a phase I trial with chronic myeloid leukemia patients
    • Peng B, Hayes M, Resta D, et al. Pharmacokinetics and pharmacodynamics of imatinib in a phase I trial with chronic myeloid leukemia patients. J Clin Oncol 2004; 22(5): 935-942.
    • (2004) J Clin Oncol , vol.22 , Issue.5 , pp. 935-942
    • Peng, B.1    Hayes, M.2    Resta, D.3
  • 7
    • 19444376971 scopus 로고    scopus 로고
    • Drug metabolism and variability among patients in drug response
    • Wilkinson GR. Drug metabolism and variability among patients in drug response. N Engl J Med 2005; 352(21): 2211-2221.
    • (2005) N Engl J Med , vol.352 , Issue.21 , pp. 2211-2221
    • Wilkinson, G.R.1
  • 8
    • 24344482346 scopus 로고    scopus 로고
    • Clinical pharmacokinetics of imatinib
    • Peng B, Lloyd P, Schran H. Clinical pharmacokinetics of imatinib. Clin Pharmacokinet 2005; 44(9): 879-94.
    • (2005) Clin Pharmacokinet , vol.44 , Issue.9 , pp. 879-894
    • Peng, B.1    Lloyd, P.2    Schran, H.3
  • 9
    • 52649177063 scopus 로고    scopus 로고
    • Multidrug resistance gene (MDR1) polymorphisms are associated with major molecular responses to standard-dose imatinib in chronic myeloid leukemia
    • Dulucq S, Bouchet S, Turcq B, et al. Multidrug resistance gene (MDR1) polymorphisms are associated with major molecular responses to standard-dose imatinib in chronic myeloid leukemia. Blood 2008; 112(5): 2024-2027.
    • (2008) Blood , vol.112 , Issue.5 , pp. 2024-2027
    • Dulucq, S.1    Bouchet, S.2    Turcq, B.3
  • 10
    • 77954936879 scopus 로고    scopus 로고
    • Functional activity of the OCT-1 protein is predictive of long-term outcome in patients with chronic-phase chronic myeloid leukemia treated with imatinib
    • White DL, Dang P, Engler J, et al. Functional activity of the OCT-1 protein is predictive of long-term outcome in patients with chronic-phase chronic myeloid leukemia treated with imatinib. J Clin Oncol 2010; 28(16): 2761-2767.
    • (2010) J Clin Oncol , vol.28 , Issue.16 , pp. 2761-2767
    • White, D.L.1    Dang, P.2    Engler, J.3
  • 11
    • 68549084355 scopus 로고    scopus 로고
    • CML patients with low OCT-1 activity achieve better molecular responses on high dose imatinib than on standard dose. those with high OCT-1 activity have excellent responses on either dose: a TOPS correlative study
    • abstract 3187].
    • White DL, Saunders VA, Dang P. CML patients with low OCT-1 activity achieve better molecular responses on high dose imatinib than on standard dose. those with high OCT-1 activity have excellent responses on either dose: a TOPS correlative study. Blood 2008; 112(11): 1093-1094[abstract 3187].
    • (2008) Blood , vol.112 , Issue.11 , pp. 1093-1094
    • White, D.L.1    Saunders, V.A.2    Dang, P.3
  • 12
    • 33745085275 scopus 로고    scopus 로고
    • OCT-1-mediated influx is a key determinant of the intracellular uptake of imatinib but not nilotinib (AMN107): reduced OCT-1 activity is the cause of low in vitro sensitivity to imatinib
    • White DL, Saunders VA, Dang P, et al. OCT-1-mediated influx is a key determinant of the intracellular uptake of imatinib but not nilotinib (AMN107): reduced OCT-1 activity is the cause of low in vitro sensitivity to imatinib. Blood 2006; 108(2): 697-704.
    • (2006) Blood , vol.108 , Issue.2 , pp. 697-704
    • White, D.L.1    Saunders, V.A.2    Dang, P.3
  • 13
    • 79952179437 scopus 로고    scopus 로고
    • Drug interactions with the tyrosine kinase inhibitors imatinib, dasatinib, and nilotinib
    • Haouala A, Widmer N, Duchosal MA, et al. Drug interactions with the tyrosine kinase inhibitors imatinib, dasatinib, and nilotinib. Blood 2011; 117(8): e75-87.
    • (2011) Blood , vol.117 , Issue.8
    • Haouala, A.1    Widmer, N.2    Duchosal, M.A.3
  • 14
    • 33745698739 scopus 로고    scopus 로고
    • Disposition of imatinib and its metabolite CGP74588 in a patient with chronic myelogenous leukemia and short-bowel syndrome
    • Beumer JH, Natale JJ, Lagattuta TF, et al. Disposition of imatinib and its metabolite CGP74588 in a patient with chronic myelogenous leukemia and short-bowel syndrome. Pharmacotherapy 2006; 26(7): 903-907.
    • (2006) Pharmacotherapy , vol.26 , Issue.7 , pp. 903-907
    • Beumer, J.H.1    Natale, J.J.2    Lagattuta, T.F.3
  • 15
    • 43249083718 scopus 로고    scopus 로고
    • Imatinib pharmacokinetics and its correlation with response and safety in chronic-phase chronic myeloid leukemia: a subanalysis of the IRIS study
    • Larson RA, Druker BJ, Guilhot FA, et al. Imatinib pharmacokinetics and its correlation with response and safety in chronic-phase chronic myeloid leukemia: a subanalysis of the IRIS study. Blood 2008; 111(8): 4022-4028.
    • (2008) Blood , vol.111 , Issue.8 , pp. 4022-4028
    • Larson, R.A.1    Druker, B.J.2    Guilhot, F.A.3
  • 16
    • 34147174980 scopus 로고    scopus 로고
    • Trough imatinib plasma levels are associated with both cytogenetic and molecular responses to standard-dose imatinib in chronic myeloid leukemia
    • Picard S, Titier K, Etienne G, et al. Trough imatinib plasma levels are associated with both cytogenetic and molecular responses to standard-dose imatinib in chronic myeloid leukemia. Blood 2007; 109(8): 3496-3499.
    • (2007) Blood , vol.109 , Issue.8 , pp. 3496-3499
    • Picard, S.1    Titier, K.2    Etienne, G.3
  • 17
    • 71849114712 scopus 로고    scopus 로고
    • Therapeutic drug monitoring of imatinib and impact on clinical decision making
    • abstract 4820].
    • Blasdel C, Wang Y, Lagattuta T, et al. Therapeutic drug monitoring of imatinib and impact on clinical decision making. Blood 2006; 108(11): 290b[abstract 4820].
    • (2006) Blood , vol.108 , Issue.11
    • Blasdel, C.1    Wang, Y.2    Lagattuta, T.3
  • 18
    • 0038359665 scopus 로고    scopus 로고
    • Liquid chromatographic-mass spectrometric assay for quantitation of imatinib and its main metabolite (CGP 74588) in plasma
    • Parise RA, Ramanathan RK, Hayes MJ, et al. Liquid chromatographic-mass spectrometric assay for quantitation of imatinib and its main metabolite (CGP 74588) in plasma. J Chromatogr B Analyt Technol Biomed Life Sci. 2003; 791(1-2): 39-44.
    • (2003) J Chromatogr B Analyt Technol Biomed Life Sci. , vol.791 , Issue.1-2 , pp. 39-44
    • Parise, R.A.1    Ramanathan, R.K.2    Hayes, M.J.3
  • 19
    • 37049028176 scopus 로고    scopus 로고
    • Most CML patients who have a suboptimal response to imatinib have low OCT-1 activity: higher doses of imatinib may overcome the negative impact of low OCT-1 activity
    • White DL, Saunders VA, Dang P, et al. Most CML patients who have a suboptimal response to imatinib have low OCT-1 activity: higher doses of imatinib may overcome the negative impact of low OCT-1 activity. Blood 2007; 110(12): 4064-4072.
    • (2007) Blood , vol.110 , Issue.12 , pp. 4064-4072
    • White, D.L.1    Saunders, V.A.2    Dang, P.3
  • 20
    • 9444282642 scopus 로고    scopus 로고
    • Active transport of imatinib into and out of cells: implications for drug resistance
    • Thomas J, Wang L, Clark RE, et al. Active transport of imatinib into and out of cells: implications for drug resistance. Blood 2004; 104(12): 3739-3745.
    • (2004) Blood , vol.104 , Issue.12 , pp. 3739-3745
    • Thomas, J.1    Wang, L.2    Clark, R.E.3
  • 21
    • 77953723830 scopus 로고    scopus 로고
    • 24months update of the TOPS study: a phase III, randomized, open-label study of 400mg/d (SD-IM) versus 800mg/d (HD-IM) of imatinib mesylate (IM) in patients (pts) with newly diagnosed, previously untreated chronic myeloid leukemia in chronic phase (CML-CP)
    • abstract 337].
    • Baccarani M, Druker BJ, Cortes-Franco J, et al. 24months update of the TOPS study: a phase III, randomized, open-label study of 400mg/d (SD-IM) versus 800mg/d (HD-IM) of imatinib mesylate (IM) in patients (pts) with newly diagnosed, previously untreated chronic myeloid leukemia in chronic phase (CML-CP). Blood 2009; 114(22): 142[abstract 337].
    • (2009) Blood , vol.114 , Issue.22 , pp. 142
    • Baccarani, M.1    Druker, B.J.2    Cortes-Franco, J.3
  • 22
    • 52449100460 scopus 로고    scopus 로고
    • Dasatinib cellular uptake and efflux in chronic myeloid leukemia cells: therapeutic implications
    • Hiwase DK, Saunders V, Hewett D, et al. Dasatinib cellular uptake and efflux in chronic myeloid leukemia cells: therapeutic implications. Clin Cancer Res 2008; 14(12): 3881-3888.
    • (2008) Clin Cancer Res , vol.14 , Issue.12 , pp. 3881-3888
    • Hiwase, D.K.1    Saunders, V.2    Hewett, D.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.