Trafficking defects in WASH-knockout fibroblasts originate from collapsed endosomal and lysosomal networks.

Molecular biology of the cell

Volumn 23, Issue 16, 2012, Pages 3215-3228

  Gomez, Timothy S ^a   Gorman, Jacquelyn A ^b   de Narvajas, Amaia Artal Martinez ^b   Koenig, Alexander O ^b   Billadeau, Daniel D ^b  

^a MAYO GRADUATE SCHOOL   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ACTIN; ACTIN BINDING PROTEIN; EARLY ENDOSOME ANTIGEN 1; EPIDERMAL GROWTH FACTOR RECEPTOR; MULTIPROTEIN COMPLEX; TRANSFERRIN RECEPTOR; VESICULAR TRANSPORT PROTEIN; WASH PROTEIN, MOUSE;

ANIMAL; ARTICLE; CELL CULTURE; CELL SURVIVAL; ENDOSOME; FIBROBLAST; FLUORESCENCE MICROSCOPY; GENE INACTIVATION; GENETICS; HUMAN; LYSOSOME; METABOLISM; MOUSE; MOUSE MUTANT; PHYSIOLOGY; PROTEIN DEGRADATION; PROTEIN TRANSPORT; ULTRASTRUCTURE;

ACTINS; ANIMALS; CELL SURVIVAL; CELLS, CULTURED; ENDOSOMES; FIBROBLASTS; GENE KNOCKOUT TECHNIQUES; HUMANS; LYSOSOMES; MICE; MICE, KNOCKOUT; MICROFILAMENT PROTEINS; MICROSCOPY, FLUORESCENCE; MULTIPROTEIN COMPLEXES; PROTEIN TRANSPORT; PROTEOLYSIS; RECEPTOR, EPIDERMAL GROWTH FACTOR; RECEPTORS, TRANSFERRIN; VESICULAR TRANSPORT PROTEINS;

EID: 84870520072     PISSN: None     EISSN: 19394586     Source Type: Journal    
DOI: 10.1091/mbc.e12-02-0101     Document Type: Article

References (0)