Anti-inflammatoryeffect and mechanism of artemisinin and dihydroartemisinin

Zhongguo Zhongyao Zazhi

Volumn 37, Issue 17, 2012, Pages 2618-2621

  Yu, Wan Ying ^a   Kan, Wei Juan ^a   Yu, Peng Xia ^a   Li, Min Min ^a   Song, Ji Shuai ^b   Zhao, Feng ^a  

^a YANTAI UNIVERSITY   (China)

^b Yantai Science and Biotechnology Co Ltd   (China)

Author keywords

Artemisinin; COX 2; Dihydroartemisinin; IL 6; NO; TNF

Indexed keywords

ARTEMISININ; BETA ACTIN; CYCLOOXYGENASE 2; DIHYDROARTEMISININ; INDUCIBLE NITRIC OXIDE SYNTHASE; INTERLEUKIN 6; LIPOPOLYSACCHARIDE; NITRIC OXIDE; TUMOR NECROSIS FACTOR ALPHA; ANTIINFLAMMATORY AGENT; ARTEMISININ DERIVATIVE; AUTACOID;

ANIMAL CELL; ANTIINFLAMMATORY ACTIVITY; ARTICLE; CYTOTOXICITY; DRUG MECHANISM; ENZYME LINKED IMMUNOSORBENT ASSAY; MACROPHAGE; MOUSE; NONHUMAN; ANIMAL; CELL LINE; DRUG EFFECT; GENE EXPRESSION; GENETICS; IMMUNOLOGY;

ANIMALS; ANTI-INFLAMMATORY AGENTS; ARTEMISININS; CELL LINE; GENE EXPRESSION; INFLAMMATION MEDIATORS; INTERLEUKIN-6; MACROPHAGES; MICE; NITRIC OXIDE; TUMOR NECROSIS FACTOR-ALPHA;

EID: 84870161990     PISSN: 10015302     EISSN: None     Source Type: Journal    
DOI: 10.4268/cjcmm20121723     Document Type: Article

References (7)

1. Farrell A J, Blake D R, Palmer R M, et al. Increased concentrations of nitrite in synovial fluid and serum samples suggest increased nitric oxide synthesis in rheumatic diseases[J]. Ann Rheum Dis, 1992,51(11):1219.