Nicotine, food intake, and activation of POMC neurons

Neuropsychopharmacology

Volumn 38, Issue 1, 2013, Pages 245-

  Picciotto, Marina R ^a   Mineur, Yann S ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACETYLCHOLINE; NICOTINE; NICOTINIC RECEPTOR; NICOTINIC RECEPTOR BETA2; NICOTINIC RECEPTOR BETA4; PROOPIOMELANOCORTIN; UNCLASSIFIED DRUG;

ACETYLCHOLINE RELEASE; FOOD INTAKE; GENE FUNCTION; HUMAN; LIFE EXPECTANCY; NERVE CONDUCTION; NONHUMAN; NOTE; OBESITY; PRIORITY JOURNAL; SMOKING; TOBACCO DEPENDENCE;

ANIMALS; EATING; HUMANS; NEURONS; NICOTINE; PRO-OPIOMELANOCORTIN; SMOKING;

EID: 84869041420     PISSN: 0893133X     EISSN: 1740634X     Source Type: Journal    
DOI: 10.1038/npp.2012.163     Document Type: Note

References (6)

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2. Huang H, Xu Y, van den Pol A (2011). Nicotine excites hypothalamic arcuate anorexigenic proopiomelanocortin neurons and orexigenic neuropeptide Y neurons: similarities and differences. J Neurophysiol 106: 1191-1202.
3. Mineur YS, Abizaid A, Rao Y, Salas R, DiLeone RJ, Gundisch D et al (2011). Nicotine decreases food intake through activation of POMC neurons. Science 332: 1330-1332.
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