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Volumn 18, Issue 9, 2012, Pages 1340-1341
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A sweet spot to control complement-induced inflammation
a
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Author keywords
[No Author keywords available]
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Indexed keywords
COMPLEMENT;
COMPLEMENT COMPONENT C5A;
COMPLEMENT COMPONENT C5A RECEPTOR;
DECTIN 1;
FC RECEPTOR;
IMMUNOGLOBULIN;
IMMUNOGLOBULIN G;
PROTEIN TYROSINE PHOSPHATASE SHP;
C5AR1 PROTEIN, HUMAN;
COMPLEMENT RECEPTOR;
LECTIN;
ADAPTIVE IMMUNITY;
ANTIGEN ANTIBODY COMPLEX;
ANTIINFLAMMATORY ACTIVITY;
ARTICLE;
ASTHMA;
AUTOIMMUNE DISEASE;
CELL ADHESION;
CELL DAMAGE;
COMPLEMENT ACTIVATION;
DEGENERATIVE DISEASE;
DRUG EFFICACY;
GOLGI COMPLEX;
GRAFT REJECTION;
HISTOPATHOLOGY;
HUMAN;
IMMUNOCOMPETENT CELL;
IMMUNOMODULATION;
IN VITRO STUDY;
IN VIVO STUDY;
INFLAMMATION;
INNATE IMMUNITY;
MEDIATOR;
MOLECULAR RECOGNITION;
NEOPLASM;
NEUTROPHIL;
NONHUMAN;
PATHOPHYSIOLOGY;
PERIODONTAL DISEASE;
PHAGOCYTOSIS;
PRIORITY JOURNAL;
PROTEIN BINDING;
PROTEIN FUNCTION;
PROTEIN PHOSPHORYLATION;
PROTEIN SECRETION;
PROTEIN SYNTHESIS;
RHEUMATOID ARTHRITIS;
SEPSIS;
SIGNAL TRANSDUCTION;
ANIMAL;
FEMALE;
IMMUNOLOGY;
METABOLISM;
NOTE;
ANIMALS;
AUTOIMMUNE DISEASES;
COMPLEMENT C5A;
FEMALE;
IMMUNOGLOBULIN G;
LECTINS, C-TYPE;
RECEPTORS, COMPLEMENT;
RECEPTORS, IGG;
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EID: 84868661963
PISSN: 10788956
EISSN: 1546170X
Source Type: Journal
DOI: 10.1038/nm.2916 Document Type: Article |
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Times cited : (7)
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References (11)
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