-
1
-
-
0025756673
-
Hyperhomocysteinemia: An independent risk factor for vascular disease
-
Clarke R, Daly L, Robinson K, Naughten E, Cahalane S, Fowler B et al. Hyperhomocysteinemia: an independent risk factor for vascular disease. N Engl J Med 1991;324: 1149-1155.
-
(1991)
N Engl J Med
, vol.324
, pp. 1149-1155
-
-
Clarke, R.1
Daly, L.2
Robinson, K.3
Naughten, E.4
Cahalane, S.5
Fowler, B.6
-
2
-
-
0037164104
-
Homocysteine and risk of ischemic heart disease and stroke: A meta-analysis
-
Homocysteine and risk of ischemic heart disease and stroke: a meta-analysis. JAMA 2002;288:2015-2022.
-
(2002)
JAMA
, vol.288
, pp. 2015-2022
-
-
-
3
-
-
0037216677
-
ADMA and oxidative stress are responsible for endothelial dysfunction in hyperhomocyst (e)inemia: Effects of L-arginine and B vitamins
-
Sydow K, Schwedhelm E, Arakawa N, Bode-Boger SM, Tsikas D, Hornig B et al. ADMA and oxidative stress are responsible for endothelial dysfunction in hyperhomocyst (e)inemia: effects of L-arginine and B vitamins. Cardiovasc Res 2003;57: 244-252.
-
(2003)
Cardiovasc Res
, vol.57
, pp. 244-252
-
-
Sydow, K.1
Schwedhelm, E.2
Arakawa, N.3
Bode-Boger, S.M.4
Tsikas, D.5
Hornig, B.6
-
4
-
-
0032771346
-
Homocysteine metabolism
-
Selhub J. Homocysteine metabolism. Annu Rev Nutr 1999;19:217-246.
-
(1999)
Annu Rev Nutr
, vol.19
, pp. 217-246
-
-
Selhub, J.1
-
5
-
-
79954621298
-
Role of sulfur-containing gaseous substances in the cardiovascular system
-
Hart JL. Role of sulfur-containing gaseous substances in the cardiovascular system. Front Biosci (Elite Ed) 2011;3:736-749.
-
(2011)
Front Biosci (Elite Ed)
, vol.3
, pp. 736-749
-
-
Hart, J.L.1
-
6
-
-
0035109723
-
Elevation of asymmetrical dimethylarginine may mediate endothelial dysfunction during experimental hyperhomocyst(e)inaemia in humans
-
Boger RH, Lentz SR, Bode-Boger SM, Knapp HR, Haynes WG. Elevation of asymmetrical dimethylarginine may mediate endothelial dysfunction during experimental hyperhomocyst(e)inaemia in humans. Clin Sci (Lond) 2001;100:161-167.
-
(2001)
Clin Sci (Lond)
, vol.100
, pp. 161-167
-
-
Boger, R.H.1
Lentz, S.R.2
Bode-Boger, S.M.3
Knapp, H.R.4
Haynes, W.G.5
-
7
-
-
71749111560
-
Cancer incidence and mortality after treatment with folic acid and vitamin B12
-
Ebbing M, Bonaa KH, Nygard O, Arnesen E, Ueland PM, Nordrehaug JE et al. Cancer incidence and mortality after treatment with folic acid and vitamin B12. JAMA 2009; 302:2119-2126.
-
(2009)
JAMA
, vol.302
, pp. 2119-2126
-
-
Ebbing, M.1
Bonaa, K.H.2
Nygard, O.3
Arnesen, E.4
Ueland, P.M.5
Nordrehaug, J.E.6
-
8
-
-
84862794123
-
Systemic and vascular oxidation limits the efficacy of oral tetrahydrobiopterin treatment in patients with coronary artery disease
-
Cunnington C, Van AT, Shirodaria C, Kylintireas I, Lindsay AC, Lee JM et al. Systemic and vascular oxidation limits the efficacy of oral tetrahydrobiopterin treatment in patients with coronary artery disease. Circulation 2012;125:1356-1366.
-
(2012)
Circulation
, vol.125
, pp. 1356-1366
-
-
Cunnington, C.1
Van At Shirodaria, C.2
Kylintireas, I.3
Lindsay, A.C.4
Lee, J.M.5
-
9
-
-
0029124027
-
Supplementation of hypercholesterolaemic rabbits with L-arginine reduces the vascular release of superoxide anions and restores NO production
-
Boger RH, Bode-Boger SM, Mugge A, Kienke S, Brandes R, Dwenger A et al. Supplementation of hypercholesterolaemic rabbits with L-arginine reduces the vascular release of superoxide anions and restores NO production. Atherosclerosis 1995; 117:273-284.
-
(1995)
Atherosclerosis
, vol.117
, pp. 273-284
-
-
Boger, R.H.1
Bode-Boger, S.M.2
Mugge, A.3
Kienke, S.4
Brandes, R.5
Dwenger, A.6
-
10
-
-
33846611102
-
Low-dose L-arginine administration increases microperfusion of hindlimb muscle without affecting blood pressure in rats
-
Ohta F, Takagi T, Sato H, Ignarro LJ. Low-dose L-arginine administration increases microperfusion of hindlimb muscle without affecting blood pressure in rats. Proc Natl Acad Sci USA 2007;104:1407-1411.
-
(2007)
Proc Natl Acad Sci USA
, vol.104
, pp. 1407-1411
-
-
Ohta, F.1
Takagi, T.2
Sato, H.3
Ignarro, L.J.4
-
11
-
-
34447334222
-
L-Arginine supplementation in peripheral arterial disease: No benefit and possible harm
-
Wilson AM, Harada R, Nair N, Balasubramanian N, Cooke JP. L-Arginine supplementation in peripheral arterial disease: no benefit and possible harm. Circulation 2007; 116:188-195.
-
(2007)
Circulation
, vol.116
, pp. 188-195
-
-
Wilson, A.M.1
Harada, R.2
Nair, N.3
Balasubramanian, N.4
Cooke, J.P.5
-
12
-
-
46849095596
-
Regulation of eNOS-derived superoxide by endogenous methylarginines
-
Druhan LJ, Forbes SP, Pope AJ, Chen CA, Zweier JL, Cardounel AJ. Regulation of eNOS-derived superoxide by endogenous methylarginines. Biochemistry 2008;47: 7256-7263.
-
(2008)
Biochemistry
, vol.47
, pp. 7256-7263
-
-
Druhan, L.J.1
Forbes, S.P.2
Pope, A.J.3
Chen, C.A.4
Zweier, J.L.5
Cardounel, A.J.6
-
13
-
-
0343551110
-
Arginase activity in endothelial cells: Inhibition by NG-hydroxy-L-arginine during high-output NO production
-
Buga GM, Singh R, Pervin S, Rogers NE, Schmitz DA, Jenkinson CP et al. Arginase activity in endothelial cells: inhibition by NG-hydroxy-L-arginine during high-output NO production. Am J Physiol 1996;271:H1988-H1998.
-
(1996)
Am J Physiol
, vol.271
-
-
Buga, G.M.1
Singh, R.2
Pervin, S.3
Rogers, N.E.4
Schmitz, D.A.5
Jenkinson, C.P.6
-
14
-
-
78650810596
-
S-glutathionylation uncouples eNOS and regulates its cellular and vascular function
-
Chen CA, Wang TY, Varadharaj S, Reyes LA, Hemann C, Talukder MA et al. S-glutathionylation uncouples eNOS and regulates its cellular and vascular function. Nature 2010;468:1115-1118.
-
(2010)
Nature
, vol.468
, pp. 1115-1118
-
-
Chen, C.A.1
Wang, T.Y.2
Varadharaj, S.3
Reyes, L.A.4
Hemann, C.5
Talukder, M.A.6
-
15
-
-
64749090979
-
ENOS activation by physical forces: From short-term regulation of contraction to chronic remodeling of cardiovascular tissues
-
Balligand JL, Feron O, Dessy C. eNOS activation by physical forces: from short-term regulation of contraction to chronic remodeling of cardiovascular tissues. Physiol Rev 2009;89:481-534.
-
(2009)
Physiol Rev
, vol.89
, pp. 481-534
-
-
Balligand, J.L.1
Feron, O.2
Dessy, C.3
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