Lys(203) and Lys(382) are essential for the proteasomal degradation of BACE1.

Current Alzheimer research

Volumn 9, Issue 5, 2012, Pages 606-615

  Wang, Rong ^a   Ying, Zhengxin ^b   Zhao, Juan ^b   Zhang, Yanyan ^b   Wang, Ran ^b   Lu, Hong ^b   Deng, Yulin ^b   Song, Weihong ^b   Qing, Hong ^b  

^a BEIJING INSTITUTE OF TECHNOLOGY   (China)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; ASPARTIC PROTEINASE; BACE1 PROTEIN, HUMAN; COMPLEMENTARY DNA; LYSINE; SECRETASE;

ALZHEIMER DISEASE; ARTICLE; CELL STRAIN HEK293; CODON; ENZYMOLOGY; GENETICS; HUMAN; METABOLISM; PHYSIOLOGY; SITE DIRECTED MUTAGENESIS; UBIQUITINATION;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; AMYLOID PRECURSOR PROTEIN SECRETASES; ASPARTIC ACID ENDOPEPTIDASES; CODON; DNA, COMPLEMENTARY; HEK293 CELLS; HUMANS; LYSINE; MUTAGENESIS, SITE-DIRECTED; UBIQUITINATION;

EID: 84866491262     PISSN: None     EISSN: 18755828     Source Type: Journal    
DOI: 10.2174/156720512800618026     Document Type: Article

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