Differences in iNOS and arginase expression and activity in the macrophages of rats are responsible for the resistance against T. gondii infection.

PloS one

Volumn 7, Issue 4, 2012, Pages

  Li, Zhi ^a   Zhao, Zhi Jun ^b   Zhu, Xing Quan ^b   Ren, Qing Shi ^b   Nie, Fang Fang ^b   Gao, Jiang Mei ^b   Gao, Xiao Jie ^b   Yang, Ting Bao ^b   Zhou, Wen Liang ^b   Shen, Ji Long ^b   Wang, Yong ^b   Lu, Fang Li ^b   Chen, Xiao Guang ^b   Hide, Geoff ^b   Ayala, Francisco J ^b   Lun, Zhao Rong ^b  

^a SUN YAT SEN UNIVERSITY   (China)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ARGINASE; INDUCIBLE NITRIC OXIDE SYNTHASE; NITRIC OXIDE;

ANIMAL; ARTICLE; BIOSYNTHESIS; DISEASE RESISTANCE; ENZYMOLOGY; GENE EXPRESSION; GENETICS; HOST RANGE; HUMAN; IMMUNOLOGY; MACROPHAGE ACTIVATION; METABOLISM; MOUSE; MOUSE STRAIN; PARASITOLOGY; PERITONEUM MACROPHAGE; PHYSIOLOGY; RAT; RAT STRAIN; RODENT DISEASE; TOXOPLASMA; TOXOPLASMOSIS;

ANIMALS; ARGINASE; DISEASE RESISTANCE; GENE EXPRESSION; HOST SPECIFICITY; HUMANS; MACROPHAGE ACTIVATION; MACROPHAGES, PERITONEAL; MICE; MICE, INBRED STRAINS; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE II; RATS; RATS, INBRED STRAINS; RODENT DISEASES; TOXOPLASMA; TOXOPLASMOSIS, ANIMAL;

EID: 84866148221     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0035834     Document Type: Article

References (0)