Arginylation regulates myofibrils to maintain heart function and prevent dilated cardiomyopathy

Journal of Molecular and Cellular Cardiology

Volumn 53, Issue 3, 2012, Pages 333-341

  Kurosaka, Satoshi ^a,f   Leu, N Adrian ^a   Pavlov, Ivan ^b   Han, Xuemei ^c   Ribeiro, Paula Aver Bretanha ^d   Xu, Tao ^c   Bunte, Ralph ^e   Saha, Sougata ^a,g   Wang, Junling ^a   Cornachione, Anabelle ^b   Mai, Wilfried ^a   Yates, John R ^c   Rassier, Dilson E ^b   Kashina, Anna ^a  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

^b MCGILL UNIVERSITY   (Canada)

^c SCRIPPS RESEARCH INSTITUTE   (United States)

^d FEDERAL UNIVERSITY OF RIO GRANDE DO SUL   (Brazil)

^e DUKE NUS MEDICAL SCHOOL   (Singapore)

^f HIROSHIMA UNIVERSITY   (Japan)

^g TEZPUR UNIVERSITY   (India)

Author keywords

Arginylation; Dilated cardyomyopathy; Myofibrils

Indexed keywords

ALPHA ACTININ 2; ALPHA TROPOMYOSIN; ALPHA1 TROPOMYOSIN; ARGINYLTRANSFERASE; FIBRILLIN 1; LAMININ GAMMA1; MYOSIN HEAVY CHAIN; MYOSIN HEAVY CHAIN ALPHA; MYOSIN HEAVY POLYPEPTIDE 6; MYOSIN LIGHT CHAIN; MYOSIN LIGHT POLYPEPTIDE 3; TROPONIN T; TROPONIN T2; UNCLASSIFIED DRUG;

ADULT ANIMAL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONGESTIVE CARDIOMYOPATHY; CONTROLLED STUDY; HEART DEVELOPMENT; HEART FUNCTION; HEART MUSCLE CONTRACTILITY; IN VIVO STUDY; LETHALITY; MOUSE; MUSCLE FIBRIL; NEWBORN; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN ARGINYLATION; PROTEIN MODIFICATION; SARCOMERE; THROMBOSIS; ULTRASTRUCTURE;

AMINOACYLTRANSFERASES; ANIMALS; CARDIOMYOPATHY, DILATED; GENES, LETHAL; HEART; MICE; MICE, KNOCKOUT; MYOCARDIAL CONTRACTION; MYOCARDIUM; MYOFIBRILS; SARCOMERES;

MUS;

EID: 84864817775     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2012.05.007     Document Type: Article

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