Genetic recombination is directed away from functional genomic elements in mice

Nature

Volumn 485, Issue 7400, 2012, Pages 642-645

  Brick, Kevin ^a   Smagulova, Fatima ^b   Khil, Pavel ^a   Camerini Otero, R Daniel ^a   Petukhova, Galina V ^b  

^a NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES   (United States)

^b UNIFORMED SERVICES UNIVERSITY OF THE HEALTH SCIENCES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALLELE; ENZYME ACTIVITY; GAMETOGENESIS; GENETIC ANALYSIS; GENOMICS; MAMMAL; RECOMBINATION; RODENT;

ALLELE; ARTICLE; FUNCTIONAL GENOMICS; GENE INACTIVATION; GENE MAPPING; GENETIC ANALYSIS; GENETIC ASSOCIATION; GENETIC RECOMBINATION; GENOME; MOUSE; NONHUMAN; PRIORITY JOURNAL; AMINO ACID SEQUENCE; ANIMAL; CHEMISTRY; DOUBLE STRANDED DNA BREAK; GENETICS; MEIOSIS; METABOLISM; METHYLATION; MOLECULAR GENETICS; MOUSE MUTANT; NUCLEOTIDE SEQUENCE; PROMOTER REGION;

MAMMALIA; MUS;

HISTONE; HISTONE LYSINE METHYLTRANSFERASE; PRDM9 PROTEIN, MOUSE;

ALLELES; AMINO ACID SEQUENCE; ANIMALS; BASE SEQUENCE; DNA BREAKS, DOUBLE-STRANDED; GENOME; HISTONE-LYSINE N-METHYLTRANSFERASE; HISTONES; MEIOSIS; METHYLATION; MICE; MICE, KNOCKOUT; MOLECULAR SEQUENCE DATA; PROMOTER REGIONS, GENETIC; RECOMBINATION, GENETIC;

EID: 84863770992     PISSN: 00280836     EISSN: 14764687     Source Type: Journal    
DOI: 10.1038/nature11089     Document Type: Article

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