Redundant Notch1 and Notch2 signaling is necessary for IFNγ secretion by T helper 1 cells during infection with Leishmania major.

PLoS pathogens

Volumn 8, Issue 3, 2012, Pages

  Auderset, Floriane ^a   Schuster, Steffen ^b   Coutaz, Manuel ^b   Koch, Ute ^b   Desgranges, Florian ^b   Merck, Estelle ^b   MacDonald, H Robson ^b   Radtke, Freddy ^b   Tacchini Cottier, Fabienne ^b  

^a UNIVERSITY OF LAUSANNE   (Switzerland)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE; GAMMA INTERFERON; NOTCH1 PROTEIN, MOUSE; NOTCH1 RECEPTOR; NOTCH2 PROTEIN, MOUSE; NOTCH2 RECEPTOR;

ANIMAL; ARTICLE; C57BL MOUSE; DISEASE MODEL; GENE EXPRESSION; GENETICS; HOST PARASITE INTERACTION; IMMUNOLOGY; LEISHMANIA MAJOR; METABOLISM; MOUSE; PHYSIOLOGY; SECRETION; SIGNAL TRANSDUCTION; SKIN LEISHMANIASIS; TH1 CELL; TRANSGENIC MOUSE;

ANIMALS; CYTOKINES; DISEASE MODELS, ANIMAL; GENE EXPRESSION; HOST-PARASITE INTERACTIONS; INTERFERON-GAMMA; LEISHMANIA MAJOR; LEISHMANIASIS, CUTANEOUS; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; RECEPTOR, NOTCH1; RECEPTOR, NOTCH2; SIGNAL TRANSDUCTION; TH1 CELLS;

EID: 84863598267     PISSN: None     EISSN: 15537374     Source Type: Journal    
DOI: 10.1371/journal.ppat.1002560     Document Type: Article

References (0)