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Volumn 21, Issue 7, 2012, Pages 554-556

Mouse Samd9l is not a functional paralogue of the human SAMD9, the gene mutated in normophosphataemic familial tumoral calcinosis

(8) Li, Qiaoli a Guo, Haitao a Matsui, Hirotaka b Honda, Hiroaki b Inaba, Toshiya b Sundberg, John P c Sprecher, Eli d Uitto, Jouni a

a THOMAS JEFFERSON UNIVERSITY (United States)

b HIROSHIMA UNIVERSITY (Japan)

c JACKSON LABORATORY (United States)

d TEL AVIV SOURASKY MEDICAL CENTER (Israel)

Author keywords

Ectopic connective tissue mineralization; Familial tumoral calcinosis; Mouse models of skin diseases; Pseudoxanthoma elasticum

Indexed keywords

PROTEIN; SAMD9 LIKE PROTEIN; SAMD9 PROTEIN; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; AUTOPSY; CHROMOSOME 7Q; FAMILIAL DISEASE; GENE DELETION; GENE LOCATION; GENE MUTATION; GENE REARRANGEMENT; KNOCKOUT MOUSE; LETTER; MINERALIZATION; MOUSE; NONHUMAN; NORMOPHOSPHATEMIC FAMILIAL TUMOR CALCINOSIS; PSEUDOXANTHOMA ELASTICUM; TUMOR CALCINOSIS;

ANIMALS; CALCINOSIS; HETEROZYGOTE; HOMOZYGOTE; HUMANS; MICE; MICE, KNOCKOUT; PHOSPHATES; PROTEINS; SKIN DISEASES; TUMOR SUPPRESSOR PROTEINS;

EID: 84863083802 PISSN: 09066705 EISSN: 16000625 Source Type: Journal
DOI: 10.1111/j.1600-0625.2012.01524.x Document Type: Letter

Times cited : (3)

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