Endoplasmic reticulum stress-induced JNK activation is a critical event leading to mitochondria-mediated cell death caused by β-lapachone treatment.

PloS one

Volumn 6, Issue 6, 2011, Pages

  Lee, Hyemi ^a   Park, Moon Taek ^b   Choi, Bo Hwa ^b   Oh, Eun Taex ^b   Song, Min Jeong ^b   Lee, Jeonghun ^b   Kim, Chulhee ^b   Lim, Byung Uk ^b   Park, Heon Joo ^b  

^a Inha University School of Medicine   (South Korea)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

BETA LAPACHONE; BETA-LAPACHONE; NAPHTHOQUINONE; SMALL INTERFERING RNA; STRESS ACTIVATED PROTEIN KINASE;

ARTICLE; CELL DEATH; CONFOCAL MICROSCOPY; DRUG EFFECT; ENDOPLASMIC RETICULUM STRESS; GENETICS; HUMAN; METABOLISM; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; PHYSIOLOGY; TUMOR CELL LINE; WESTERN BLOTTING;

BLOTTING, WESTERN; CELL DEATH; CELL LINE, TUMOR; ENDOPLASMIC RETICULUM STRESS; HUMANS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICROSCOPY, CONFOCAL; MITOCHONDRIA; NAPHTHOQUINONES; RNA, SMALL INTERFERING;

EID: 84862833343     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0021533     Document Type: Article

References (0)