High-glucose environment reduces human β-defensin-2 expression in human keratinocytes: Implications for poor diabetic wound healing

British Journal of Dermatology

Volumn 166, Issue 6, 2012, Pages 1221-1229

  Lan, C C E ^a,b,c   Wu, C S ^d   Huang, S M ^a,b,e   Kuo, H Y ^a,b   Wu, I H ^a,b   Liang, C W ^a,b   Chen, G S ^a,b  

^a KAOHSIUNG MEDICAL UNIVERSITY HOSPITAL   (Taiwan)

^b KAOHSIUNG MEDICAL UNIVERSITY   (Taiwan)

^c KAOHSIUNG MUNICIPAL TA TUNG HOSPITAL   (Taiwan)

^d KAOHSIUNG MEDICAL UNIVERSITY   (Taiwan)

^e NATIONAL SUN YAT SEN UNIVERSITY   (Taiwan)

Author keywords

[No Author keywords available]

Indexed keywords

ADVANCED GLYCATION END PRODUCT; BETA DEFENSIN 2; GLUCOSE; INTERLEUKIN 1BETA; MESSENGER RNA; SMALL INTERFERING RNA; STAT1 PROTEIN;

ARTICLE; CELL VIABILITY; CONCENTRATION (PARAMETERS); CONTROLLED STUDY; DIABETES MELLITUS; DOWN REGULATION; GENE SILENCING; GENETIC TRANSCRIPTION; HUMAN; HUMAN CELL; HUMAN CELL CULTURE; HUMAN EXPERIMENT; KERATINOCYTE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION; UPREGULATION; WOUND HEALING;

BETA-DEFENSINS; BLOTTING, WESTERN; CELL SURVIVAL; DIABETES MELLITUS; GENE SILENCING; GLUCOSE; HUMANS; KERATINOCYTES; RNA, MESSENGER; RNA, SMALL INTERFERING; STAT1 TRANSCRIPTION FACTOR; WOUND HEALING;

EID: 84861608410     PISSN: 00070963     EISSN: 13652133     Source Type: Journal    
DOI: 10.1111/j.1365-2133.2012.10847.x     Document Type: Article

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