α-1-antitrypsin gene delivery reduces inflammation, increases T-regulatory cell population size and prevents islet allograft rejection.

Molecular medicine (Cambridge, Mass.)

Volumn 17, Issue 9-10, 2011, Pages 1000-1011

  Shahaf, Galit ^a   Moser, Hadas ^b   Ozeri, Eyal ^b   Mizrahi, Mark ^b   Abecassis, Avishag ^b   Lewis, Eli C ^b  

^a BEN GURION UNIVERSITY OF THE NEGEV   (Israel)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA 1 ANTITRYPSIN; ANTISERUM; LIPOPOLYSACCHARIDE;

ALLOTRANSPLANTATION; ANIMAL; ARTICLE; C57BL MOUSE; CELL LINE; CYTOLOGY; DRUG EFFECT; GENE THERAPY; GENE TRANSFER; GENETICS; GRAFT REJECTION; HUMAN; IMMUNOLOGY; INFLAMMATION; LYMPHOCYTE COUNT; MACROPHAGE; METABOLISM; METHODOLOGY; MOUSE; PANCREAS ISLET TRANSPLANTATION; PLASMID; REGULATORY T LYMPHOCYTE; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; WESTERN BLOTTING;

ALPHA 1-ANTITRYPSIN; ANIMALS; BLOTTING, WESTERN; CELL LINE; GENE THERAPY; GENE TRANSFER TECHNIQUES; GRAFT REJECTION; HUMANS; IMMUNE SERA; INFLAMMATION; ISLETS OF LANGERHANS TRANSPLANTATION; LIPOPOLYSACCHARIDES; LYMPHOCYTE COUNT; MACROPHAGES; MICE; MICE, INBRED C57BL; PLASMIDS; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; T-LYMPHOCYTES, REGULATORY; TRANSPLANTATION, HOMOLOGOUS;

EID: 84858776868     PISSN: None     EISSN: 15283658     Source Type: Journal    
DOI: 10.2119/molmed.2011.00145     Document Type: Article

References (0)