Adaptive responses to alloxan-induced mild oxidative stress ameliorate certain tauopathy phenotypes

Aging Cell

Volumn 11, Issue 1, 2012, Pages 51-62

  Yoshiike, Yuji ^a,b   Yamashita, Shunji ^a   Mizoroki, Tatsuya ^a   Maeda, Sumihiro ^c   Murayama, Miyuki ^a   Kimura, Tetsuya ^a,b   Sahara, Naruhiko ^d   Soeda, Yoshiyuki ^a   Takashima, Akihiko ^a,b  

^a RIKEN BRAIN SCIENCE INSTITUTE   (Japan)

^b NATIONAL CENTER FOR GERIATRICS AND GERONTOLOGY   (Japan)

^c UNIVERSITY OF CALIFORNIA   (United States)

^d UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

Author keywords

Aging; Alzheimer's disease; Memory; Microtubule; Oxidative stress; Tau

Indexed keywords

ALLOXAN; CELL CYCLE PROTEIN 37; GLUCOSE; HISTONE DEACETYLASE 2; HISTONE DEACETYLASE 6; PROTEASOME; STRESS ACTIVATED PROTEIN KINASE; TAU PROTEIN; TUBULIN;

ACETYLATION; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; CONTROLLED STUDY; FEMALE; GLUCOSE BLOOD LEVEL; HUMAN; IN VIVO STUDY; MALE; MEMORY; MICROTUBULE; MOUSE; NERVE CELL; NONHUMAN; OXIDATIVE STRESS; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; TAUOPATHY;

ACETYLATION; ADAPTATION, PHYSIOLOGICAL; AGING; ALLOXAN; ANIMALS; DISEASE MODELS, ANIMAL; FEMALE; HISTONE DEACETYLASES; HSP90 HEAT-SHOCK PROTEINS; HUMANS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MALE; MAZE LEARNING; MICE; MICE, TRANSGENIC; NEURONS; OXIDATIVE STRESS; PHENOTYPE; PHOSPHORYLATION; PROTEASOME ENDOPEPTIDASE COMPLEX; TAU PROTEINS; TAUOPATHIES; TUBULIN;

MURINAE; MUS; MUS MUSCULUS;

EID: 84855851105     PISSN: 14749718     EISSN: 14749726     Source Type: Journal    
DOI: 10.1111/j.1474-9726.2011.00756.x     Document Type: Article

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