Myogenic progenitors contribute to open but not closed fracture repair.

BMC musculoskeletal disorders

Volumn 12, Issue , 2011, Pages 288-

  Liu, Renjing ^a   Birke, Oliver ^b   Morse, Alyson ^b   Peacock, Lauren ^b   Mikulec, Kathy ^b   Little, David G ^b   Schindeler, Aaron ^b  

^a CHILDREN S HOSPITAL AT WESTMEAD   (Australia)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ALKALINE PHOSPHATASE; CRE RECOMBINASE; INTEGRASE; MYOD PROTEIN;

129 MOUSE; ANIMAL; ARTICLE; C57BL MOUSE; CARTILAGE CELL; CELL LINEAGE; CELL TRANSDIFFERENTIATION; DISEASE MODEL; FRACTURE; FRACTURE HEALING; GENETICS; HUMAN; INJURY; METABOLISM; MOUSE; OPEN FRACTURE; OSTEOBLAST; PATHOLOGY; PROMOTER REGION; REPORTER GENE; SKELETAL MUSCLE SATELLITE CELL; STEM CELL; TIBIA; TIBIA FRACTURE; TIME; TRANSGENIC MOUSE;

ALKALINE PHOSPHATASE; ANIMALS; CELL LINEAGE; CELL TRANSDIFFERENTIATION; CHONDROCYTES; DISEASE MODELS, ANIMAL; FRACTURE HEALING; FRACTURES, CLOSED; FRACTURES, OPEN; GENES, REPORTER; HUMANS; INTEGRASES; MICE; MICE, 129 STRAIN; MICE, INBRED C57BL; MICE, TRANSGENIC; MYOD PROTEIN; OSTEOBLASTS; PROMOTER REGIONS, GENETIC; SATELLITE CELLS, SKELETAL MUSCLE; STEM CELLS; TIBIA; TIBIAL FRACTURES; TIME FACTORS;

MLCS; MLOWN;

EID: 84055213708     PISSN: None     EISSN: 14712474     Source Type: Journal    
DOI: 10.1186/1471-2474-12-288     Document Type: Article

References (0)