Effects of a reduction in the number of gap junction channels or in their conductance on ischemia-reperfusion arrhythmias in isolated mouse hearts

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 301, Issue 6, 2011, Pages 2442-2453

  Sánchez, Jose A ^a   Rodríguez Sinovas, Antonio ^a   Fernández Sanz, Celia ^a   Ruiz Meana, Marisol ^a   García Dorado, David ^a  

^a HOSPITAL UNIVERSITARI VALL D HEBRON   (Spain)

Author keywords

Connexin43; Electrical impedance

Indexed keywords

AFIMOXIFENE; CONNEXIN 32; CONNEXIN 43;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARRHYTHMOGENESIS; ARTICLE; CARDIOVASCULAR RISK; CELL INTERACTION; CONTROLLED STUDY; DRUG ACTIVITY; ELECTROCHEMICAL IMPEDANCE SPECTROSCOPY; ELECTROPHYSIOLOGY; FEMALE; GAP JUNCTION; HEART MUSCLE; HEART VENTRICLE ARRHYTHMIA; ISCHEMIA; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; REPERFUSION INJURY;

ACTION POTENTIALS; ANIMALS; ARRHYTHMIAS, CARDIAC; CONNEXIN 43; CONNEXINS; DISEASE MODELS, ANIMAL; DOWN-REGULATION; ELECTRIC IMPEDANCE; FEMALE; GAP JUNCTIONS; HEART VENTRICLES; MALE; MICE; MICE, 129 STRAIN; MICE, INBRED C57BL; MICE, TRANSGENIC; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; PERFUSION; PERMEABILITY; TIME FACTORS;

EID: 82855169269     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00540.2011     Document Type: Article

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