Role of endogenous ROS production in impaired metabolism-secretion coupling of diabetic pancreatic β cells

Progress in Biophysics and Molecular Biology

Volumn 107, Issue 2, 2011, Pages 304-310

  Fujimoto, Shimpei ^a   Mukai, Eri ^a   Inagaki, Nobuya ^a  

^a KYOTO UNIVERSITY   (Japan)

Author keywords

ATP; Insulin secretion; Pancreatic cell; Reactive oxygen species; Src

Indexed keywords

GLUCAGON LIKE PEPTIDE RECEPTOR; GLUCAGON RECEPTOR; GLUCAGON-LIKE PEPTIDE RECEPTOR; PROTEIN TYROSINE KINASE; REACTIVE OXYGEN METABOLITE;

ANIMAL; ARTICLE; DIABETES MELLITUS; DRUG EFFECT; DRUG POTENTIATION; ENZYME ACTIVATION; ENZYMOLOGY; HUMAN; METABOLISM; PANCREAS ISLET BETA CELL; PATHOLOGY; SECRETION;

ANIMALS; DIABETES MELLITUS; ENZYME ACTIVATION; HUMANS; INSULIN-SECRETING CELLS; REACTIVE OXYGEN SPECIES; RECEPTORS, GLUCAGON; SRC-FAMILY KINASES;

RATTUS;

EID: 82455210858     PISSN: 00796107     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.pbiomolbio.2011.07.013     Document Type: Article

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