Dysfunctional γ T cells contribute to impaired keratinocyte homeostasis in mouse models of obesity

Journal of Investigative Dermatology

Volumn 131, Issue 12, 2011, Pages 2409-2418

  Taylor, Kristen R ^a   Costanzo, Anne E ^a   Jameson, Julie M ^a  

^a SCRIPPS RESEARCH INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

UVOMORULIN; CADHERIN; LYMPHOCYTE ANTIGEN RECEPTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL ADHESION; CELL FUNCTION; CONTROLLED STUDY; EPIDERMIS; EPITHELIUM; FEMALE; GAMMA DELTA T LYMPHOCYTE; HOMEOSTASIS; KERATINOCYTE; MALE; MOUSE; NONHUMAN; OBESITY; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; SKIN DEFECT; SKIN PERMEABILITY; SKIN STRUCTURE; ANIMAL; C57BL MOUSE; DISEASE MODEL; METABOLISM; MOUSE MUTANT; NON INSULIN DEPENDENT DIABETES MELLITUS; PATHOPHYSIOLOGY; PHYSIOLOGY; T LYMPHOCYTE SUBPOPULATION;

ANIMALS; CADHERINS; DIABETES MELLITUS, TYPE 2; DISEASE MODELS, ANIMAL; FEMALE; HOMEOSTASIS; KERATINOCYTES; MALE; MICE; MICE, INBRED C57BL; MICE, OBESE; OBESITY; RECEPTORS, ANTIGEN, T-CELL, GAMMA-DELTA; T-LYMPHOCYTE SUBSETS;

EID: 80855130834     PISSN: 0022202X     EISSN: 15231747     Source Type: Journal    
DOI: 10.1038/jid.2011.241     Document Type: Article

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