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Volumn 124, Issue 19, 2011, Pages 2052-2055

Diversity of myocardial interstitial proteolytic pathways: Gene deletion reveals unexpected consequences

Author keywords

[No Author keywords available]

Indexed keywords

GELATINASE A; TISSUE INHIBITOR OF METALLOPROTEINASE 2;

EID: 80855128850     PISSN: 00097322     EISSN: 15244539     Source Type: Journal    
DOI: 10.1161/CIRCULATIONAHA.111.064071     Document Type: Review
Times cited : (8)

References (12)
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  • 3
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    • Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress
    • Kandalam V, Basu R, Moore L, Fan D, Wang X, Jaworski DM, Oudit GY, Kassiri Z. Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress. Circulation. 2011;124: 2094-2105.
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  • 4
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    • Matrix metalloproteinases: What do they not do? new substrates and biological roles identified by murine models and proteomics
    • Rodriquez D, Morrison CH, Overall CM. Matrix metalloproteinases: what do they not do? new substrates and biological roles identified by murine models and proteomics. Biochem Biophys Acta. 2010;1803: 39-54.
    • (2010) Biochem Biophys Acta. , vol.1803 , pp. 39-54
    • Rodriquez, D.1    Morrison, C.H.2    Overall, C.M.3
  • 5
    • 79951778377 scopus 로고    scopus 로고
    • Myocardial remodeling: Cellular and extracellular events and targets
    • Dixon JA, Spinale FG. Myocardial remodeling: cellular and extracellular events and targets. Annu Rev Physiol. 2011;73:47-68.
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    • Dixon, J.A.1    Spinale, F.G.2
  • 7
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    • Cardiac restricted over-expression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarction
    • Spinale FG, Mukherjee R, Zavadzkas JA, Koval CN, Bouges S, Stroud RE, Dobrucki LW, Sinusas AJ. Cardiac restricted over-expression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarction. J Biol Chem. 2010;285: 30316-30327.
    • (2010) J Biol Chem , vol.285 , pp. 30316-30327
    • Spinale, F.G.1    Mukherjee, R.2    Zavadzkas, J.A.3    Koval, C.N.4    Bouges, S.5    Stroud, R.E.6    Dobrucki, L.W.7    Sinusas, A.J.8
  • 8
    • 35748974863 scopus 로고    scopus 로고
    • Myocardial matrix remodeling and the matrix metalloproteinases: Influence on cardiac form and function
    • DOI 10.1152/physrev.00012.2007
    • Spinale FG. Matrix remodeling and the matrix metalloproteinases: influence on cardiac form and function. Physiol Rev. 2007;87:1285-1342. (Pubitemid 350041472)
    • (2007) Physiological Reviews , vol.87 , Issue.4 , pp. 1285-1342
    • Spinale, F.G.1
  • 9
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    • The tissue inhibitors of metalloproteinases (TIMPs): An ancient family with structural and functional diversity
    • Brew K, Nagase H. The tissue inhibitors of metalloproteinases (TIMPs): an ancient family with structural and functional diversity. Biochim et Biophys Acta. 2010:1803;55-71.
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    • Brew, K.1    Nagase, H.2
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    • Macrophage-specific metalloelastase (MMP-12) truncates and inactivates ELR-CXC chemokines and generates CCL2,-7,-8 and-13 antagonists: Potential role of the macrophage in terminating polymorphonuclear leukocyte influx
    • Dean RA, Cox JH, Bellac CL, Doucet A, Starr AE, Overall CM. Macrophage-specific metalloelastase (MMP-12) truncates and inactivates ELR-CXC chemokines and generates CCL2,-7,-8 and-13 antagonists: potential role of the macrophage in terminating polymorphonuclear leukocyte influx. Blood. 2008;112:3455-3464.
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    • Plasma biomarkers that reflect determinants of matrix composition identify the presence of left ventricular hypertrophy and diastolic heart failure
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.