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Volumn 797, Issue , 2012, Pages 53-63

Syrian hamster tumor model to study oncolytic ad5-based vectors

Author keywords

Cancer gene therapy; Immunosuppression; Intraperitoneal tumors; Neutralization; Oncolytic adenovirus vector; Syrian hamster model

Indexed keywords

ADENOVIRUS; ANIMAL; ARTICLE; EXPERIMENTAL NEOPLASM; GENE VECTOR; GENETICS; HAMSTER; HUMAN; METHODOLOGY; ONCOLYTIC VIROTHERAPY; SYRIAN HAMSTER; VIRUS REPLICATION;

EID: 80555127437     PISSN: 10643745     EISSN: 19406029     Source Type: Book Series    
DOI: 10.1007/978-1-61779-340-0_4     Document Type: Chapter
Times cited : (13)

References (15)
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  • 2
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    • Effect of preexisting immunity on oncolytic adenovirus vector INGN 007 antitumor efficacy in immunocompetent and immunosuppressed Syrian hamsters
    • Dhar, D., Spencer, J. F., Toth, K., and Wold, W. S. (2009) Effect of preexisting immunity on oncolytic adenovirus vector INGN 007 antitumor efficacy in immunocompetent and immunosuppressed Syrian hamsters. J. Virol. 83, 2130–9
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    • Dhar, D.1    Spencer, J. F.2    Toth, K.3    Wold, W. S.4
  • 3
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    • Pre-existing Immunity and Passive Immunity to Adenovirus 5 Prevents Toxicity Caused by an Oncolytic Adenovirus Vector in the Syrian Hamster Model
    • Dhar, D., Spencer, J. F., Toth, K., and Wold, W. S. (2009) Pre-existing Immunity and Passive Immunity to Adenovirus 5 Prevents Toxicity Caused by an Oncolytic Adenovirus Vector in the Syrian Hamster Model. Mol. Ther. 17, 1724–32
    • (2009) Mol. Ther , vol.17 , pp. 1724-1732
    • Dhar, D.1    Spencer, J. F.2    Toth, K.3    Wold, W. S.4
  • 4
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    • An acute toxicology study with INGN 007, an oncolytic adenovirus vector, in mice and permissive Syrian hamsters; comparisons with wild-type Ad5 and a replication-defective adenovirus vector
    • Lichtenstein, D. L., Spencer, J. F., Doronin, K., Patra, D., Meyer, J. M., Shashkova, E. V., et al. (2009) An acute toxicology study with INGN 007, an oncolytic adenovirus vector, in mice and permissive Syrian hamsters; comparisons with wild-type Ad5 and a replication-defective adenovirus vector. Cancer Gene Ther. 16, 644–54
    • (2009) Cancer Gene Ther , vol.16 , pp. 644-654
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  • 5
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    • INGN 007, an oncolytic adenovirus vector, replicates in Syrian hamsters but not mice: comparison of biodistribution studies
    • Ying, B., Toth, K., Spencer, J. F., Meyer, J., Tollefson, A. E., Patra, D., et al. (2009) INGN 007, an oncolytic adenovirus vector, replicates in Syrian hamsters but not mice: comparison of biodistribution studies. Cancer Gene Ther. 16, 625–37
    • (2009) Cancer Gene Ther , vol.16 , pp. 625-637
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  • 6
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    • New pancreatic carcinoma model for studying oncolytic adenoviruses in the permissive Syrian hamster
    • Spencer, J. F., Sagartz, J. E., Wold, W. S. M., and Toth, K. (2009) New pancreatic carcinoma model for studying oncolytic adenoviruses in the permissive Syrian hamster. Cancer Gene Ther. 16, 912–22
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  • 7
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    • Thomas, M. A., Spencer, J. F., Toth, K., Sagartz, J. E., Phillips, N. J., and Wold, W. S. (2008) Immunosuppression enhances oncolytic adenovirus replication and antitumor efficacy in the Syrian hamster model. Mol. Ther. 16, 1665–73
    • (2008) Mol. Ther , vol.16 , pp. 1665-1673
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  • 8
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    • Hexadecyloxypropyl-cidofovir, CMX001, prevents adenovirus-induced mortality in a permissive, immunosuppressed animal model
    • Toth, K., Spencer, J. F., Dhar, D., Sagartz, J. E., Buller, R. M., Painter, G. R., et al. (2008) Hexadecyloxypropyl-cidofovir, CMX001, prevents adenovirus-induced mortality in a permissive, immunosuppressed animal model. Proc. Natl. Acad. Sci.USA 105, 7293–7
    • (2008) Proc. Natl. Acad. Sci.USA , vol.105 , pp. 7293-7297
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  • 10
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    • Deletion of the E3-6.7K/gp19K region reduces the persistence of wild-type adenovirus in a permissive tumor model in Syrian hamsters
    • Bortolanza, S., Bunuales, M., Alzuguren, P., Lamas, O., Aldabe, R., Prieto, J., et al. (2009) Deletion of the E3-6.7K/gp19K region reduces the persistence of wild-type adenovirus in a permissive tumor model in Syrian hamsters. Cancer Gene Ther. 16, 703–12
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.