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Volumn 202, Issue 5, 2011, Pages 553-557

Vascular smooth muscle cell migration induced by domains of thrombospondin-1 is differentially regulated

Author keywords

Domains; Fragments; Migration; Thrombospondin; Vascular smooth muscle cells

Indexed keywords

2 (2 AMINO 3 METHOXYPHENYL)CHROMONE; 2 MORPHOLINO 8 PHENYLCHROMONE; 4 (1 AMINOETHYL) N (4 PYRIDYL)CYCLOHEXANECARBOXAMIDE; 4 (4 FLUOROPHENYL) 2 (4 HYDROXYPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; PROCOLLAGEN; RECOMBINANT PROTEIN; THROMBOSPONDIN 1;

EID: 80055014590     PISSN: 00029610     EISSN: 18791883     Source Type: Journal    
DOI: 10.1016/j.amjsurg.2011.06.025     Document Type: Article
Times cited : (6)

References (8)
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  • 2
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  • 3
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    • Thrombospondin 1, fibronectin, and vitronectin are differentially dependent upon RAS, ERK1/2, and p38 for induction of vascular smooth muscle cell chemotaxis
    • A.I. Willis, B. Sadowitz, and S. Fuse Thrombospondin 1, fibronectin, and vitronectin are differentially dependent upon RAS, ERK1/2, and p38 for induction of vascular smooth muscle cell chemotaxis Vasc Endovascular Surg 45 2011 55 62
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    • The C-terminal domain of thrombospondin-1 induces vascular smooth muscle cell chemotaxis
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    • Thrombospondin-1 type 1 repeat recombinant proteins inhibit tumor growth through transforming growth factor-beta-dependent and -independent mechanisms
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    • Thrombospondin-1-induced vascular smooth muscle cell chemotaxis: The role of the type 3 repeat and carboxyl terminal domains
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    • Thrombospondin induces RhoA inactivation through FAK-dependent signaling to stimulate focal adhesion disassembly
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.