Response to comment on "The inhibiting Fc receptor for IgG, FcγRIIB, is a modifier of autoimmune susceptibility"

Journal of Immunology

Volumn 187, Issue 8, 2011, Pages 3909-3910

  Verbeek, Sjef ^a   Boross, Peter ^a   Izui, Shozo ^b  

^a LEIDEN UNIVERSITY MEDICAL CENTER   (Netherlands)

^b UNIVERSITY OF GENEVA   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIBODY; FC RECEPTOR; FC RECEPTOR IIB; IMMUNOGLOBULIN G;

AUTOIMMUNE DISEASE; AUTOIMMUNITY; DISEASE PREDISPOSITION; DNA FLANKING REGION; FCGR 2B GENE; GENE; GENE FUNCTION; GENETIC VARIABILITY; HAPLOTYPE; IFI202 GENE; IMMUNOGLOBULIN GENE; LETTER; NBA2 GENE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SYSTEMIC LUPUS ERYTHEMATOSUS;

EID: 80054728437     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1190056     Document Type: Letter

References (1)

1. Jørgensen, T. N., J. Alfaro, H. L. Enriquez, C. Jiang, W. M. Loo, S. Atencio, M. R. Bupp, C. M. Mailloux, T. Metzger, S. Flannery, et al. 2010. Development of murine lupus involves the combined genetic contribution of the SLAM and FcgammaR intervals within the Nba2 autoimmune susceptibility locus. J. Immunol. 184: 775-786.