DNA base excision repair gene polymorphisms modulate human cognitive performance and decline during normal life span

Mechanisms of Ageing and Development

Volumn 132, Issue 8-9, 2011, Pages 449-458

  Lillenes, Meryl S ^a   Espeseth, Thomas ^a   Støen, Mari ^b   Lundervold, Astri J ^c,d   Frye, Stephan A ^c   Rootwelt, Helge ^b   Reinvang, Ivar ^a   Tønjum, Tone ^a,b  

^a UNIVERSITY OF OSLO   (Norway)

^b OSLO UNIVERSITY HOSPITAL   (Norway)

^c UNIVERSITY OF BERGEN   (Norway)

^d STATOIL   (Norway)

Author keywords

Base excision repair; Cognitive decline; DNA repair; Normal brain aging; Oxidative DNA damage; Single nucleotide polymorphisms

Indexed keywords

ADULT; AGED; AGING; APOE GENE; ARTICLE; BER GENE; BINDING SITE; BRAIN FUNCTION; CHRNA4 GENE; COGNITION; CONTROLLED STUDY; DNA BINDING; DNA POLYMORPHISM; EPISODIC MEMORY; EXCISION REPAIR; EXON; FEMALE; GENE; GENE DELETION; GENE FREQUENCY; GENE FUNCTION; GENE INSERTION; GENOTYPE; HOMOZYGOSITY; HUMAN; MALE; NUCLEOTIDE SEQUENCE; PHENOTYPE; PRIORITY JOURNAL; SINGLE NUCLEOTIDE POLYMORPHISM; TASK PERFORMANCE; WORKING MEMORY;

ADULT; AGED; AGING; BIOLOGICAL MARKERS; COGNITION; CROSS-SECTIONAL STUDIES; DNA REPAIR; FEMALE; HUMANS; MALE; MIDDLE AGED; POLYMORPHISM, SINGLE NUCLEOTIDE;

EID: 80053107063     PISSN: 00476374     EISSN: 18726216     Source Type: Journal    
DOI: 10.1016/j.mad.2011.08.002     Document Type: Article

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