Reply to "Reversal and prevention of arsenic-induced human bronchial epithelial cell malignant transformation by microrna-200b"

Toxicological Sciences

Volumn 122, Issue 2, 2011, Pages 606-606

  Cohen, Samuel M ^a  

^a UNIVERSITY OF NEBRASKA MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ARSENIC; ARSENIC TRIOXIDE; MICRORNA; MICRORNA 200B; ORGANOARSENIC DERIVATIVE; PROTEIN P53; UNCLASSIFIED DRUG;

BRONCHUS MUCOSA; CELL DIFFERENTIATION; CELL PROLIFERATION; EPITHELIAL MESENCHYMAL TRANSITION; EPITHELIUM CELL; HISTOPATHOLOGY; HUMAN; IMMUNOHISTOCHEMISTRY; IN VITRO STUDY; LETTER; LUNG CARCINOMA; LUNG TUMOR; MALIGNANT TRANSFORMATION; MESENCHYME CELL; NONHUMAN; TUMOR GROWTH;

ANIMALS; ARSENIC; BRONCHI; CELL TRANSFORMATION, NEOPLASTIC; HUMANS; MICRORNAS;

EID: 80051688678     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kfr128     Document Type: Letter

References (2)

1. Somji, S., Zhou, X. D., Garrett, S. H., Sens, M. A., and Sens, D. A. (2006). Urothelial cells malignantly transformed by exposure to cadmium (Cd(+2)) and arsenite (As(+3)) have increased resistance to Cd(+2) and As(+3)-induced cell death. Toxicol. Sci. 94, 293-301.
2. Wnek, S. M., Jensen, T. J., Severson, P. L., Futscher, B. W., and Gandolfi, A. J. (2010). Monomethylarsonous acid produces irreversible events resulting in malignant transformation of a human bladder cell line following 12 weeks of low-level exposure. Toxicol. Sci. 116, 44-57.