Ischemic preconditioning attenuates mitochondrial localization of PTEN induced by ischemia-reperfusion

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 300, Issue 6, 2011, Pages

  Zu, Lingyun ^a,b   Zheng, Xiaoxu ^a   Wang, Bing ^a   Parajuli, Nirmal ^a   Steenbergen, Charles ^a   Becker, Lewis C ^a   Cai, Zheqing P ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b PEKING UNIVERSITY THIRD HOSPITAL   (China)

Author keywords

Apoptosis; Bax; Phosphatase and tensin homologs deleted on chromosome 10; Reperfusion injury

Indexed keywords

ACETYLCYSTEINE; HYDROGEN PEROXIDE; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE 3 PHOSPHATASE; PROTEIN BAX; REACTIVE OXYGEN METABOLITE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL SURVIVAL; CELL VIABILITY; CONTROLLED STUDY; HEART INFARCTION SIZE; HEART MUSCLE; HEART PERFUSION; HYPOTHESIS; ISCHEMIC PRECONDITIONING; ISOLATED HEART; MALE; MITOCHONDRIAL TARGETING SIGNAL; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; REPERFUSION INJURY; WILD TYPE;

ANIMALS; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; HYDROGEN PEROXIDE; ISCHEMIC PRECONDITIONING, MYOCARDIAL; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MITOCHONDRIA, HEART; MODELS, ANIMAL; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; PTEN PHOSPHOHYDROLASE; REACTIVE OXYGEN SPECIES;

EID: 79958073565     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.01138.2010     Document Type: Article

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