Tissue vibration induces carotid artery endothelial dysfunction: A mechanism linking snoring and carotid atherosclerosis?

Sleep

Volumn 34, Issue 6, 2011, Pages 751-757

  Cho, Jin Gun ^a,b   Witting, Paul K ^b   Verma, Manisha ^a   Wu, Ben J ^c   Shanu, Anu ^b   Kairaitis, Kristina ^a,b   Amis, Terence C ^a,b   Wheatley, John R ^a,b  

^a WESTMEAD HOSPITAL   (Australia)

^b UNIVERSITY OF SYDNEY   (Australia)

^c HEART RESEARCH INSTITUTE   (Australia)

Author keywords

Carotid atherosclerosis; Endothelial dysfunction; Snoring; Vibration

Indexed keywords

ACETYLCHOLINE; CYCLIC GMP; NITROPRUSSIDE SODIUM;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTERY DILATATION; ARTERY ENDOTHELIUM; ARTICLE; BLOOD PRESSURE; CAROTID ARTERY; CAROTID ATHEROSCLEROSIS; CONTROLLED STUDY; ENDOTHELIAL DYSFUNCTION; HYPOXEMIA; MALE; NONHUMAN; PRIORITY JOURNAL; SNORING; VIBRATION; WHOLE BODY VIBRATION;

ANIMALS; CAROTID ARTERY DISEASES; CELL SURVIVAL; CYCLIC GMP; DISEASE MODELS, ANIMAL; ENDOTHELIUM, VASCULAR; MALE; RABBITS; SNORING; VIBRATION;

EID: 79957935981     PISSN: 01618105     EISSN: 15509109     Source Type: Journal    
DOI: 10.5665/SLEEP.1042     Document Type: Article

References (5)

1. Amatoury J, Howitt L, Wheatley JR, Avolio AP, Amis TC. Snoring-related energy transmission to the carotid artery in rabbits. J Appl Physiol 2006;100:1547-53.
2. Tulis DA. Histological and morphometric analyses for rat carotid balloon injury model. In: Sreejayan N, Totowa RJ, eds. Methods in molecular medicine, Vol 139: Vascular biology protocols. NJ: Humana Press, 2007.
3. Witting PK, Rayner BS, Wu BJ, Ellis NA, Stocker R. Hydrogen peroxide promotes endothelial dysfunction by stimulating multiple sources of superoxide anion radical production and decreasing nitric oxide bioavailability. Cell Physiol Biochem 2007;20:255-68. (Pubitemid 47340589)
4. Smith PK, Krohn RI, Hermanson GT, et al. Measurement of protein using bicinchoninic acid. Anal Biochem 1985;150:76-85. (Pubitemid 16258399)
5. Gavrieli Y, Sherman Y, Ben-Sasson SA. Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation. J Cell Biol 1992;119:493-501.