Association study between GNB1L and three major mental disorders in Chinese Han populations

Psychiatry Research

Volumn 187, Issue 3, 2011, Pages 457-459

  Li, You ^a   Zhao, Qian ^a   Wang, Ti ^a   Liu, Jie ^a   Li, Junyan ^a   Li, Tao ^a   Zeng, Zhen ^a   Feng, Guoyin ^b   He, Lin ^a,c,d   Shi, Yongyong ^a  

^a SHANGHAI JIAO TONG UNIVERSITY   (China)

^b SHANGHAI MENTAL HEALTH CENTER   (China)

^c Chinese Academy of Sciences   (China)

^d FUDAN UNIVERSITY   (China)

Author keywords

Bipolar Disorder; Major Depressive Disorder; Schizophrenia

Indexed keywords

ADULT; ALLELE; ARTICLE; BIPOLAR DISORDER; CAUCASIAN; CHINESE; CONTROLLED STUDY; FEMALE; GENE; GENETIC ANALYSIS; GENETIC ASSOCIATION; GENOTYPE; GNB1L GENE; HUMAN; MAJOR CLINICAL STUDY; MAJOR DEPRESSION; MALE; PRIORITY JOURNAL; SCHIZOPHRENIA; SINGLE NUCLEOTIDE POLYMORPHISM;

ADULT; BIPOLAR DISORDER; CHINA; DEPRESSIVE DISORDER, MAJOR; FEMALE; GENE FREQUENCY; GENOME-WIDE ASSOCIATION STUDY; GENOTYPE; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MALE; MENTAL DISORDERS; POLYMORPHISM, SINGLE NUCLEOTIDE; SCHIZOPHRENIA;

EID: 79954610782     PISSN: 01651781     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.psychres.2010.04.019     Document Type: Article

References (13)

1. Bassett A.S., Hodgkinson K., Chow E.W., Correia S., Scutt L.E. 22q11 deletion syndrome in adults with schizophrenia. Journal of the American Psychiatric Association 1998, 81(4):328-337.
2. Blazer D.G., Kessler R.C., McGonagle K.A., Swartz M.S. The prevalence and distribution of major depressive disorder in a national community sample: the National Comorbidity Survey. Journal of the American Psychiatric Association 1994, 151:979-986.
3. Garcia-Higuera I., Fenoglio J., Li Y., Lewis C., Panchenko M.P., Reiner O., Smith T.F., Neer E.J. Folding of proteins with WD-repeats: comparison of six members of the WD-repeat superfamily to the G protein beta subunit. Biochemistry 1996, 35:13985-13994.
4. Gong L., Liu M., Jen J., Yeh T.H. GNB1L a gene deleted in the critical region for DiGeorge syndrome on 22q11, encodes a G-protein β-subunit-like polypeptide. Biochimica et Biophysica Acta 2000, 1494:185-188.
5. Ishiguro H., Koga M., Horiuchi Y., Noguchi E., Morikawa M., Suzuki Y., Arai M., Niizato K., Iritani S., Itokawa M., Inada T., Iwata N., Ozaki N., Ujike H., Kunugi H., Sasaki T., Takahashi M., Watanabe Y., Someya T., Kakita A., Takahashi H., Nawa H., Arinami T. Supportive evidence for reduced expression of GNB1L in schizophrenia. Schizophrenia Bulletin 2010, 36(4):756-765.
6. Lichtenstein P., Yip B.H., Björk C., Pawitan Y., Cannon T.D., Sullivan P.F., Hultman C.M. Common genetic determinants of schizophrenia and bipolar disorder in Swedish families: a population-based study. Lancet. 2009, 373(9659):234-239. Jan 17.
7. Maynard T.M., Haskell G.T., Peters A.Z., Sikich L., Lieberman J.A., LaMantia A.S. A comprehensive analysis of 22q11 gene expression in the developing and adult brain. Proceedings of the National Academy of Sciences of the United States of America 2003, 100:14433-14438.
8. Merikangas K.R., Akiskal H.S., Angst J., Greenberg P.E., Hirschfeld R.M., Petukhova M., Kessler R.C. Lifetime and 12-month prevalence of bipolar spectrum disorder in the National Comorbidity Survey replication. Archives of General Psychiatry 2007, 64(5):543-552.
9. Saha S., Chant D., Welham J., McGrath J. A systematic review of the prevalence of schizophrenia. PLoS Medicine 2005, 2(5):e141.
10. Shen G.Q., Abdullah K.G., Wang Q.K. The TaqMan method for SNP genotyping. Methods in Molecular Biology 2009, 578:293-306.
11. Shi Y.Y., He L. SHEsis, a powerful software platform for analyses of linkage disequilibrium, haplotype construction, and genetic association at polymorphism loci. Cell Research 2005, 15:97-98.
12. Smith T.F., Gaitatzes C., Saxena K., Neer E.J. The WD repeat: a common architecture for diverse functions. Trends in Biochemical Sciences 1999, 24(5):181-185.
13. Williams N.M., Glaser B., Norton N., Williams H., Pierce T., Moskvina V., Monks S., Del Favero J., Goossens D., Rujescu D., Giegling I., Kirov G., Craddock N., Murphy K.C., O'Donovan M.C., Owen M.J. Strong evidence that GNB1L is associated with schizophrenia. Human Molecular Genetics 2008, 17(4).